Diabetes mellitus induced by low-dose interleukin-2

Cancer Immunol Immunother. 1996 Sep;43(1):59-62. doi: 10.1007/s002620050304.

Abstract

Interleukin-2 (IL-2) is a potent immunomodulator that has been associated with the clinical development of autoimmune disorders. However, diabetes mellitus has not been reported in patients treated with single-agent IL-2. We conducted a clinical trial of a protracted daily schedule of subcutaneously administered low-dose IL-2. A patient with advanced colorectal cancer, treated with 1.5 x 10(6) international units of IL-2 daily, developed insulin-requiring diabetes during therapy. Hyperglycemia improved during treatment interruption and recurred with reinstitution of IL-2. The diabetes in this patient developed in the context of T cell and natural killer cell expansion, and the presence of islet cell autoantibodies was documented. We postulate that, in this patient, IL-2 reversed the anergy of autoreactive T cells that had escaped clonal deletion. It is possible that prolonged daily exposure to immunomodulatory doses of IL-2 will result in the development of autoimmune phenomena not observed with other schedules of administration.

Publication types

  • Case Reports

MeSH terms

  • Adjuvants, Immunologic / adverse effects
  • Diabetes Mellitus / chemically induced*
  • Diabetes Mellitus / immunology
  • Dose-Response Relationship, Drug
  • Female
  • Humans
  • Interleukin-2 / adverse effects*
  • Lymphocyte Count / drug effects
  • Middle Aged
  • Time Factors

Substances

  • Adjuvants, Immunologic
  • Interleukin-2