Stimulation of human cytotoxic T cells with HIV-1-derived peptides presented by recombinant HLA-A2 peptide complexes

Int Immunol. 1997 Mar;9(3):451-9. doi: 10.1093/intimm/9.3.451.

Abstract

HLA-A2 heavy chain and beta 2-microglobulin were expressed in Escherichia coli, and refolded in the presence of peptides derived from HIV-1 RT and gag proteins. When recombinant HLA-A2 molecules were attached to cells lacking HLA-A2, the cells became susceptible to lysis by HLA-A2-restricted cytotoxic T lymphocyte (CTL) clones specific for peptides derived from RT and gag proteins. Limiting dilution analyses of peripheral blood mononuclear cells from HIV-1-infected individuals showed that the recombinant HLA-A2 peptide complexes covalently immobilized on microspheres stimulated the development of HLA-A2 peptide-specific CTL. Preformed HLA-peptide complexes may provide an alternative to immunization procedures that depend upon intracellular processing of antigen to elicit T cell responses.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antigen Presentation*
  • Gene Products, gag / immunology*
  • HIV Antigens / immunology*
  • HIV Reverse Transcriptase / immunology*
  • HIV-1 / immunology*
  • HLA-A2 Antigen / immunology*
  • Humans
  • Lymphocyte Activation*
  • Microspheres
  • Peptide Fragments / immunology
  • Recombinant Fusion Proteins / immunology
  • T-Lymphocytes, Cytotoxic / immunology*
  • beta 2-Microglobulin / genetics
  • beta 2-Microglobulin / immunology

Substances

  • Gene Products, gag
  • HIV Antigens
  • HLA-A2 Antigen
  • Peptide Fragments
  • Recombinant Fusion Proteins
  • beta 2-Microglobulin
  • HIV Reverse Transcriptase