Iron-bleomycin interactions with oxygen and oxygen analogues. Effects on spectra and drug activity

J Biol Chem. 1979 Nov 10;254(21):10906-12.

Abstract

Despite extensive structural dissimilarities, iron . bleomycin complexes and heme-containing oxygenases display remarkable similarities in binding oxygen antagonists and in spectral properties deriving from bound iron. Fe(II)-bleomycin reversibly forms a complex with either CO or isocyanide (lambda max = 384 and 497 nm, respectively), either of which interfere with its oxygen-dependent cleavage of DNA. A similar but paramagnetic complex forms with NO (lambda max = 470 nm; AN = 24 G). In contrast, cyanide enhances bleomycin activity against DNA. Complexes of bleomycin and FE(III), formed either by direct association or by autoxidation of the Fe(II) . bleomycin complex, exhibit indistinguishable EPR and visible spectra, which change characteristically with pH. At neutral pH, Fe(III) . bleomycin is a low spin complex (g = 2.45, 2.18, 1.89; lambda max = 365, 384 nm) and, at low pH, it is a high spin rhombic complex (geff = 9.4, 4.3; lambda max = 430 nm). These complexes are interconvertible (pK 4.3). Fe(II) . bleomycin oxidation, although reversible by spectral criteria, is accompanied by drug inactivation unless DNA is present.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Azides
  • Bleomycin*
  • Carbon Monoxide
  • Cattle
  • Chemical Phenomena
  • Chemistry
  • Cyanides
  • DNA
  • Electron Spin Resonance Spectroscopy
  • Iron*
  • Malondialdehyde
  • Nitric Oxide
  • Oxidation-Reduction
  • Oxygen*
  • Spectrophotometry

Substances

  • Azides
  • Cyanides
  • Bleomycin
  • Nitric Oxide
  • Malondialdehyde
  • Carbon Monoxide
  • DNA
  • Iron
  • Oxygen