Childhood-onset anorexia nervosa: towards identifying a biological substrate

Int J Eat Disord. 1997 Sep;22(2):159-65. doi: 10.1002/(sici)1098-108x(199709)22:2<159::aid-eat7>3.0.co;2-e.

Abstract

Objective: The etiology of anorexia nervosa is not fully understood, but is probably multifactorial, including a biological substrate. The purpose of this paper is to investigate the possible underlying biological substrate.

Method: Fifteen children and adolescents aged 8-16 years underwent regional cerebral blood blow radioisotope scans. All fulfilled DSM-IV criteria for anorexia nervosa. Three of the girls had a follow-up scan when they had regained their lost weight.

Results: Thirteen of the 15 patients had unilateral temporal lobe hypoperfusion, 8 on the left side and 5 on the right. The abnormality persisted in the 3 girls who had a follow-up scan after weight restoration.

Discussion: This is the first report of reduced regional cerebral blood flow in childhood-onset anorexia nervosa, and suggests an underlying primary functional abnormality.

MeSH terms

  • Adolescent
  • Age of Onset
  • Anorexia Nervosa / diagnostic imaging*
  • Anorexia Nervosa / etiology*
  • Anorexia Nervosa / physiopathology
  • Cerebrovascular Circulation*
  • Child
  • Female
  • Follow-Up Studies
  • Humans
  • Male
  • Temporal Lobe / blood supply*
  • Tomography, Emission-Computed, Single-Photon
  • Weight Gain