T helper 2 cytokines induce preproenkephalin mRNA expression and proenkephalin A in human peripheral blood mononuclear cells

J Neuroimmunol. 1997 Oct;79(1):91-9. doi: 10.1016/s0165-5728(97)00113-6.

Abstract

We investigated the regulatory influence of several cytokines on the expression of preproenkephalin (PPE) mRNA in human peripheral blood mononuclear cells (PBMC). By use of a quantitative reverse-transcriptase-polymerase chain reaction (RT-PCR), we demonstrate that the T helper 2 cytokines IL-4 and IL-10 are more potent in upregulating PPE mRNA expression in human PBMC than the T helper 1 cytokines IL-2 and gamma-IFN. In addition, TGF-beta is also an effective inducer of PPE mRNA. TGF-beta, IL-4 and IL-10 increase the cytoplasmatic concentration of met-enkephalin in PBMC. Secretion of met-enkephalin in the culture supernatant of IL-4- or IL-10-stimulated PBMC could not be observed, but proenkephalin A-derived met-enkephalin containing peptides could be demonstrated. IL-4 and IL-10 do not induce PPE mRNA via the same pathways. We could observe that PKA is involved in IL-4 mediated PPE mRNA induction, whereas IL-10 apparently uses another route.

MeSH terms

  • Cells, Cultured
  • Culture Media / chemistry
  • Cyclic AMP / physiology
  • Cytokines / pharmacology
  • Drug Synergism
  • Enkephalin, Methionine / metabolism
  • Enkephalins / blood*
  • Enkephalins / genetics*
  • Humans
  • Immunohistochemistry
  • Interleukin-10 / pharmacology*
  • Interleukin-4 / pharmacology*
  • Monocytes / metabolism*
  • Polymerase Chain Reaction
  • Protein Precursors / blood*
  • Protein Precursors / genetics*
  • RNA, Messenger / metabolism*
  • Transcription, Genetic

Substances

  • Culture Media
  • Cytokines
  • Enkephalins
  • Protein Precursors
  • RNA, Messenger
  • proenkephalin
  • Interleukin-10
  • Interleukin-4
  • Enkephalin, Methionine
  • preproenkephalin
  • Cyclic AMP