CK beta-11/macrophage inflammatory protein-3 beta/EBI1-ligand chemokine is an efficacious chemoattractant for T and B cells

J Immunol. 1998 Mar 1;160(5):2418-24.

Abstract

We examined the functional properties of CK beta-11/MIP-3 beta/ELC, a recently reported CC chemokine that specifically binds to a chemokine receptor, EBI1/BLR2/CCR7. CK beta-11/MIP-3 beta/ELC is distantly related to other CC and CXC chemokines in primary amino acid sequence structure. Recombinant human CK beta-11/MIP-3 beta/ELC expressed from a mammalian cell system showed potent chemotactic activity for T cells and B cells but not for granulocytes and monocytes. An optimal concentration of CK beta-11/MIP-3 beta/ELC attracted most input T cells within 3 h, a chemotactic activity comparable with that of stromal cell derived factor 1 (SDF-1), a highly efficacious CXC chemokine. CK beta-11/MIP-3 beta/ELC equally attracted naive CD45RA+ and memory type CD45RO+ T cells. CK beta-11/MIP-3 beta/ELC also strongly attracted both CD4+ and CD8+ T cells, but the attraction for CD4+ T cells was greater. CK beta-11/MIP-3 beta/ELC was also a more efficacious chemoattractant for B cells than MIP-1 alpha, a known B cell chemoattractant. CK beta-11/MIP-3 beta/ELC induced actin polymerization in lymphocytes, and chemotaxis was completely blocked by pertussis toxin showing its receptor, most likely EBI1/BLR2/CCR7, is coupled to a G(alpha i) protein. CK beta-11/MIP-3 beta/ELC induced calcium mobilization in lymphocytes, which could be desensitized by SDF-1, suggesting possible cross-regulation in their signaling. Human CK beta-11/MIP-3 beta/ELC attracted murine splenocytes suggesting functional conservation of CK beta-11/MIP-3 beta/ELC between human and mouse. The efficacy of chemoattraction by CK beta-11/MIP-3 beta/ELC and tissue expression of its mRNA suggest that CK beta-11/MIP-3 beta/ELC may be important in trafficking of T cells in thymus, and T cell and B cell migration to secondary lymphoid organs.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Actins / metabolism
  • Animals
  • B-Lymphocytes / metabolism
  • B-Lymphocytes / physiology*
  • Calcium / metabolism
  • Chemokine CCL19
  • Chemokines / physiology
  • Chemokines, CC*
  • Chemotaxis, Leukocyte / drug effects
  • Chemotaxis, Leukocyte / immunology*
  • Granulocytes / physiology
  • Humans
  • Ligands
  • Macrophage Inflammatory Proteins / physiology*
  • Mice
  • Monocytes / physiology
  • Pertussis Toxin
  • Polymers / metabolism
  • Receptors, CCR7
  • Receptors, Cell Surface / physiology*
  • Receptors, Chemokine*
  • Spleen / cytology
  • T-Lymphocytes / metabolism
  • T-Lymphocytes / physiology*
  • Virulence Factors, Bordetella / pharmacology

Substances

  • Actins
  • CCL19 protein, human
  • CCR7 protein, human
  • Ccl19 protein, mouse
  • Ccr7 protein, mouse
  • Chemokine CCL19
  • Chemokines
  • Chemokines, CC
  • Ligands
  • Macrophage Inflammatory Proteins
  • Polymers
  • Receptors, CCR7
  • Receptors, Cell Surface
  • Receptors, Chemokine
  • Virulence Factors, Bordetella
  • Pertussis Toxin
  • Calcium