Glucocorticoids: regulation of gene expression and apoptosis

J Chemother. 1998 Apr;10(2):187-91. doi: 10.1179/joc.1998.10.2.187.
No abstract available

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Apoptosis / drug effects*
  • Apoptosis / genetics
  • Cells, Cultured
  • DNA, Complementary / biosynthesis
  • Gene Expression Regulation / drug effects*
  • Glucocorticoid-Induced TNFR-Related Protein
  • Glucocorticoids / pharmacology*
  • Hybridomas / metabolism
  • Lymphocyte Activation / genetics
  • Mice
  • Molecular Sequence Data
  • RNA, Messenger / biosynthesis
  • Receptors, Antigen, T-Cell / genetics
  • Receptors, Nerve Growth Factor / biosynthesis*
  • Receptors, Nerve Growth Factor / genetics*
  • Receptors, Tumor Necrosis Factor / biosynthesis*
  • Receptors, Tumor Necrosis Factor / genetics*
  • Sequence Homology, Amino Acid
  • T-Lymphocytes / drug effects*
  • T-Lymphocytes / metabolism
  • Transfection

Substances

  • DNA, Complementary
  • Glucocorticoid-Induced TNFR-Related Protein
  • Glucocorticoids
  • RNA, Messenger
  • Receptors, Antigen, T-Cell
  • Receptors, Nerve Growth Factor
  • Receptors, Tumor Necrosis Factor
  • Tnfrsf18 protein, mouse