Abstract
The oncogene p3k, coding for a constitutively active form of phosphatidylinositol 3-kinase (PI 3-kinase; EC 2.7.1.137), strongly enhances myogenic differentiation in cultures of chicken-embryo myoblasts. It increases the size of the myotubes and induces elevated levels of the muscle-specific proteins MyoD, myosin heavy chain, creatine kinase, and desmin. Inhibition of PI 3-kinase activity with LY294002 or with dominant-negative mutants of PI 3-kinase interferes with myogenic differentiation and with the induction of muscle-specific genes. PI 3-kinase is therefore an upstream mediator for the expression of the muscle-specific genes and is both necessary and rate-limiting for the process of myogenesis.
Publication types
-
Research Support, Non-U.S. Gov't
-
Research Support, U.S. Gov't, P.H.S.
MeSH terms
-
Animals
-
Cell Cycle / drug effects
-
Cell Differentiation* / drug effects
-
Cells, Cultured
-
Chick Embryo
-
Chromones / pharmacology
-
Creatine Kinase / genetics
-
Desmin / genetics
-
Enzyme Inhibitors / pharmacology
-
Gene Expression Regulation
-
Morpholines / pharmacology
-
Muscle, Skeletal / cytology*
-
Muscle, Skeletal / enzymology
-
MyoD Protein / genetics
-
Myosin Heavy Chains / genetics
-
Oncogenes
-
Phosphatidylinositol 3-Kinases / genetics
-
Phosphatidylinositol 3-Kinases / metabolism*
Substances
-
Chromones
-
Desmin
-
Enzyme Inhibitors
-
Morpholines
-
MyoD Protein
-
2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one
-
Phosphatidylinositol 3-Kinases
-
Creatine Kinase
-
Myosin Heavy Chains