Influence of GB virus-C/hepatitis G virus infection on the long-term course of chronic hepatitis B

Liver. 1998 Oct;18(5):360-5. doi: 10.1111/j.1600-0676.1998.tb00818.x.

Abstract

Aims/background: The clinical significance of GB virus-C/hepatitis G virus (GBV-C/HGV) infection in chronic hepatitis B is not well known and its role in the outcome of liver disease was investigated.

Methods: HGV-RNA and antibody to HGV (anti-E2) were studied in 125 patients with chronic hepatitis B (41 with multiple hepatitis virus exposure), 82 asymptomatic HBsAg carriers and 103 healthy adults.

Results: In chronic hepatitis B, HGV-RNA was more frequent in patients with HDV infection and/or anti-HCV positivity than in those without (29% vs 6%, p<0.0001), mainly in drug addicts (38%). At diagnosis the overall prevalence of any marker (HGV-RNA plus anti-E2) was similar in chronic hepatitis due to HBV alone (17%), in HBsAg carriers (16%) and in healthy adults (17%) and increased to 58% in those exposed to HDV and/or HCV. During 1-11 years of follow-up, HGV infection persisted in 70% of patients with chronic hepatitis B. About 400% of HGV persistently coinfected patients underwent sustained biochemical remission, whereas continuing disease activity was observed in 80% of patients who cleared HGV-RNA.

Conclusions: In chronic HBV infection the rate of exposure to HGV is similar to that in healthy adults, except for high risk patients. Long lasting HGV coinfection or anti-E2 seroconversion did not modify the course of chronic hepatitis B.

MeSH terms

  • Adult
  • Aged
  • DNA Primers / chemistry
  • Female
  • Flaviviridae* / genetics
  • Flaviviridae* / immunology
  • Flaviviridae* / isolation & purification
  • Follow-Up Studies
  • Hepatitis Antibodies / analysis
  • Hepatitis B virus* / immunology
  • Hepatitis B virus* / isolation & purification
  • Hepatitis B, Chronic / physiopathology*
  • Hepatitis B, Chronic / virology
  • Hepatitis Delta Virus / genetics
  • Hepatitis Delta Virus / isolation & purification
  • Hepatitis, Viral, Human / physiopathology*
  • Hepatitis, Viral, Human / virology
  • Humans
  • Male
  • Middle Aged
  • Prevalence
  • RNA, Viral / analysis
  • Reverse Transcriptase Polymerase Chain Reaction
  • Viral Envelope Proteins / analysis

Substances

  • DNA Primers
  • Hepatitis Antibodies
  • RNA, Viral
  • Viral Envelope Proteins
  • glycoprotein E2, GB virus C