Peck AB - Search Results

1

An alternative perspective to the immune response in autoimmune exocrinopathy: induction of functional quiescence rather than destructive autoaggression.

Humphreys-Beher MG, Brayer J, Yamachika S, Peck AB, Jonsson R. Humphreys-Beher MG, et al. Among authors: peck ab. Scand J Immunol. 1999 Jan;49(1):7-10. doi: 10.1046/j.1365-3083.1999.00490.x. Scand J Immunol. 1999. PMID: 10023850 Free article. Review.

2

Transfer of human serum IgG to nonobese diabetic Igmu null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome.

Robinson CP, Brayer J, Yamachika S, Esch TR, Peck AB, Stewart CA, Peen E, Jonsson R, Humphreys-Beher MG. Robinson CP, et al. Among authors: peck ab. Proc Natl Acad Sci U S A. 1998 Jun 23;95(13):7538-43. doi: 10.1073/pnas.95.13.7538. Proc Natl Acad Sci U S A. 1998. PMID: 9636185 Free PMC article.

3

Salivary gland changes in the NOD mouse model for Sjögren's syndrome: is there a non-immune genetic trigger?

Humphreys-Beher MG, Yamachika S, Yamamoto H, Maeda N, Nakagawa Y, Peck AB, Robinson CP. Humphreys-Beher MG, et al. Among authors: peck ab. Eur J Morphol. 1998 Aug;36 Suppl:247-51. Eur J Morphol. 1998. PMID: 9825931

4

Excessive synthesis of matrix metalloproteinases in exocrine tissues of NOD mouse models for Sjögren's syndrome.

Yamachika S, Nanni JM, Nguyen KH, Garces L, Lowry JM, Robinson CP, Brayer J, Oxford GE, da Silveira A, Kerr M, Peck AB, Humphreys-Beher MG. Yamachika S, et al. Among authors: peck ab. J Rheumatol. 1998 Dec;25(12):2371-80. J Rheumatol. 1998. PMID: 9858432

5

The role of apoptosis in the initiation of the autoimmune response in Sjögren's syndrome.

Humphreys-Beher MG, Peck AB, Dang H, Talal N. Humphreys-Beher MG, et al. Among authors: peck ab. Clin Exp Immunol. 1999 Jun;116(3):383-7. doi: 10.1046/j.1365-2249.1999.00888.x. Clin Exp Immunol. 1999. PMID: 10361223 Free PMC article. Review. No abstract available.

6

New concepts for the development of autoimmune exocrinopathy derived from studies with the NOD mouse model.

Humphreys-Beher MG, Peck AB. Humphreys-Beher MG, et al. Among authors: peck ab. Arch Oral Biol. 1999 May;44 Suppl 1:S21-5. doi: 10.1016/s0003-9969(99)00045-x. Arch Oral Biol. 1999. PMID: 10414851 Review.

7

Aberrant proteolytic digestion of biglycan and decorin by saliva and exocrine gland lysates from the NOD mouse model for autoimmune exocrinopathy.

Yamachika S, Brayer J, Oxford GE, Peck AB, Humphreys-Beher MG. Yamachika S, et al. Among authors: peck ab. Clin Exp Rheumatol. 2000 Mar-Apr;18(2):233-40. Clin Exp Rheumatol. 2000. PMID: 10812497

8

IL-4-dependent effector phase in autoimmune exocrinopathy as defined by the NOD.IL-4-gene knockout mouse model of Sjögren's syndrome.

Brayer JB, Cha S, Nagashima H, Yasunari U, Lindberg A, Diggs S, Martinez J, Goa J, Humphreys-Beher MG, Peck AB. Brayer JB, et al. Among authors: peck ab. Scand J Immunol. 2001 Jul-Aug;54(1-2):133-40. doi: 10.1046/j.1365-3083.2001.00958.x. Scand J Immunol. 2001. PMID: 11439159 Free article.

9

Abnormal organogenesis in salivary gland development may initiate adult onset of autoimmune exocrinopathy.

Cha S, van Blockland SC, Versnel MA, Homo-Delarche F, Nagashima H, Brayer J, Peck AB, Humphreys-Beher MG. Cha S, et al. Among authors: peck ab. Exp Clin Immunogenet. 2001;18(3):143-60. doi: 10.1159/000049194. Exp Clin Immunogenet. 2001. PMID: 11549844

10

Sjögren's syndrome: immunological response underlying the disease.

Brayer JB, Humphreys-Beher MG, Peck AB. Brayer JB, et al. Among authors: peck ab. Arch Immunol Ther Exp (Warsz). 2001;49(5):353-60. Arch Immunol Ther Exp (Warsz). 2001. PMID: 11798133 Review.

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