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Platelets from a patient heterozygous for the defect of P2CYC receptors for ADP have a secretion defect despite normal thromboxane A2 production and normal granule stores: further evidence that some cases of platelet 'primary secretion defect' are heterozygous for a defect of P2CYC receptors.
Cattaneo M, Lecchi A, Lombardi R, Gachet C, Zighetti ML. Cattaneo M, et al. Among authors: lombardi r. Arterioscler Thromb Vasc Biol. 2000 Nov;20(11):E101-6. doi: 10.1161/01.atv.20.11.e101. Arterioscler Thromb Vasc Biol. 2000. PMID: 11073862
Deficiency of (33P)2MeS-ADP binding sites on platelets with secretion defect, normal granule stores and normal thromboxane A2 production. Evidence that ADP potentiates platelet secretion independently of the formation of large platelet aggregates and thromboxane A2 production.
Cattaneo M, Lombardi R, Zighetti ML, Gachet C, Ohlmann P, Cazenave JP, Mannucci PM. Cattaneo M, et al. Among authors: lombardi r. Thromb Haemost. 1997 May;77(5):986-90. Thromb Haemost. 1997. PMID: 9184415
596 results