Fodde R - Search Results

1

Cyclooxygenase-two (COX-2) modulates proliferation in aggressive fibromatosis (desmoid tumor).

Poon R, Smits R, Li C, Jagmohan-Changur S, Kong M, Cheon S, Yu C, Fodde R, Alman BA. Poon R, et al. Among authors: fodde r. Oncogene. 2001 Jan 25;20(4):451-60. doi: 10.1038/sj.onc.1204107. Oncogene. 2001. PMID: 11313976

2

Genetic deletion of receptor for hyaluronan-mediated motility (Rhamm) attenuates the formation of aggressive fibromatosis (desmoid tumor).

Tolg C, Poon R, Fodde R, Turley EA, Alman BA. Tolg C, et al. Among authors: fodde r. Oncogene. 2003 Oct 9;22(44):6873-82. doi: 10.1038/sj.onc.1206811. Oncogene. 2003. PMID: 14534534

3

Matrix metalloproteinase activity modulates tumor size, cell motility, and cell invasiveness in murine aggressive fibromatosis.

Kong Y, Poon R, Nadesan P, Di Muccio T, Fodde R, Khokha R, Alman BA. Kong Y, et al. Among authors: fodde r. Cancer Res. 2004 Aug 15;64(16):5795-803. doi: 10.1158/0008-5472.CAN-03-3112. Cancer Res. 2004. PMID: 15313922

4

A germline mutation at the extreme 3' end of the APC gene results in a severe desmoid phenotype and is associated with overexpression of beta-catenin in the desmoid tumor.

Couture J, Mitri A, Lagace R, Smits R, Berk T, Bouchard HL, Fodde R, Alman B, Bapat B. Couture J, et al. Among authors: fodde r. Clin Genet. 2000 Mar;57(3):205-12. doi: 10.1034/j.1399-0004.2000.570306.x. Clin Genet. 2000. PMID: 10782927

5

Smad4 haploinsufficiency in mouse models for intestinal cancer.

Alberici P, Jagmohan-Changur S, De Pater E, Van Der Valk M, Smits R, Hohenstein P, Fodde R. Alberici P, et al. Among authors: fodde r. Oncogene. 2006 Mar 23;25(13):1841-51. doi: 10.1038/sj.onc.1209226. Oncogene. 2006. PMID: 16288217

6

Apc1638N: a mouse model for familial adenomatous polyposis-associated desmoid tumors and cutaneous cysts.

Smits R, van der Houven van Oordt W, Luz A, Zurcher C, Jagmohan-Changur S, Breukel C, Khan PM, Fodde R. Smits R, et al. Among authors: fodde r. Gastroenterology. 1998 Feb;114(2):275-83. doi: 10.1016/s0016-5085(98)70478-0. Gastroenterology. 1998. PMID: 9453487

7

Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity.

Smits R, Hofland N, Edelmann W, Geugien M, Jagmohan-Changur S, Albuquerque C, Breukel C, Kucherlapati R, Kielman MF, Fodde R. Smits R, et al. Among authors: fodde r. Genes Chromosomes Cancer. 2000 Nov;29(3):229-39. doi: 10.1002/1098-2264(2000)9999:9999<::aid-gcc1033>3.0.co;2-r. Genes Chromosomes Cancer. 2000. PMID: 10992298

8

APC, signal transduction and genetic instability in colorectal cancer.

Fodde R, Smits R, Clevers H. Fodde R, et al. Nat Rev Cancer. 2001 Oct;1(1):55-67. doi: 10.1038/35094067. Nat Rev Cancer. 2001. PMID: 11900252 Review.

9

E-cadherin and adenomatous polyposis coli mutations are synergistic in intestinal tumor initiation in mice.

Smits R, Ruiz P, Diaz-Cano S, Luz A, Jagmohan-Changur S, Breukel C, Birchmeier C, Birchmeier W, Fodde R. Smits R, et al. Among authors: fodde r. Gastroenterology. 2000 Oct;119(4):1045-53. doi: 10.1053/gast.2000.18162. Gastroenterology. 2000. PMID: 11040191

10

Apc1638T: a mouse model delineating critical domains of the adenomatous polyposis coli protein involved in tumorigenesis and development.

Smits R, Kielman MF, Breukel C, Zurcher C, Neufeld K, Jagmohan-Changur S, Hofland N, van Dijk J, White R, Edelmann W, Kucherlapati R, Khan PM, Fodde R. Smits R, et al. Among authors: fodde r. Genes Dev. 1999 May 15;13(10):1309-21. doi: 10.1101/gad.13.10.1309. Genes Dev. 1999. PMID: 10346819 Free PMC article.

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