Ezaki J - Search Results

1

CLC-3 deficiency leads to phenotypes similar to human neuronal ceroid lipofuscinosis.

Yoshikawa M, Uchida S, Ezaki J, Rai T, Hayama A, Kobayashi K, Kida Y, Noda M, Koike M, Uchiyama Y, Marumo F, Kominami E, Sasaki S. Yoshikawa M, et al. Among authors: ezaki j. Genes Cells. 2002 Jun;7(6):597-605. doi: 10.1046/j.1365-2443.2002.00539.x. Genes Cells. 2002. PMID: 12059962 Free article.

2

The intracellular location and function of proteins of neuronal ceroid lipofuscinoses.

Ezaki J, Kominami E. Ezaki J, et al. Brain Pathol. 2004 Jan;14(1):77-85. doi: 10.1111/j.1750-3639.2004.tb00501.x. Brain Pathol. 2004. PMID: 14997940 Free PMC article. Review.

3

Characterization of Cln3p, the gene product responsible for juvenile neuronal ceroid lipofuscinosis, as a lysosomal integral membrane glycoprotein.

Ezaki J, Takeda-Ezaki M, Koike M, Ohsawa Y, Taka H, Mineki R, Murayama K, Uchiyama Y, Ueno T, Kominami E. Ezaki J, et al. J Neurochem. 2003 Dec;87(5):1296-308. doi: 10.1046/j.1471-4159.2003.02132.x. J Neurochem. 2003. PMID: 14622109 Free article.

4

Differences in expression patterns of cathepsin C/dipeptidyl peptidase I in normal, pathological and aged mouse central nervous system.

Koike M, Shibata M, Ezaki J, Peters C, Saftig P, Kominami E, Uchiyama Y. Koike M, et al. Among authors: ezaki j. Eur J Neurosci. 2013 Mar;37(5):816-30. doi: 10.1111/ejn.12096. Epub 2012 Dec 20. Eur J Neurosci. 2013. PMID: 23279039

5

Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease).

Ezaki J, Wolfe LS, Ishidoh K, Kominami E. Ezaki J, et al. Am J Med Genet. 1995 Jun 5;57(2):254-9. doi: 10.1002/ajmg.1320570229. Am J Med Genet. 1995. PMID: 7668341 Review.

6

Specific delay of degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis (Batten disease).

Ezaki J, Wolfe LS, Higuti T, Ishidoh K, Kominami E. Ezaki J, et al. J Neurochem. 1995 Feb;64(2):733-41. doi: 10.1046/j.1471-4159.1995.64020733.x. J Neurochem. 1995. PMID: 7830067

7

Molecular basis of lysosomal accumulation of subunit c of mitochondrial ATP synthase in neuronal ceroid-lipofuscinosis.

Ezaki J, Wolfe LS, Kominami E. Ezaki J, et al. J Inherit Metab Dis. 1993;16(2):296-8. doi: 10.1007/BF00710269. J Inherit Metab Dis. 1993. PMID: 8411985 Review. No abstract available.

8

New insight into lysosomal protein storage disease: delayed catabolism of ATP synthase subunit c in Batten disease.

Kominami E, Ezaki J, Wolfe LS. Kominami E, et al. Among authors: ezaki j. Neurochem Res. 1995 Nov;20(11):1305-9. doi: 10.1007/BF00992505. Neurochem Res. 1995. PMID: 8786816 Review.

9

Defect of proteolysis of mitochondrial ATP synthase subunit C in neuronal ceroid lipofuscinosis.

Ezaki J, Wolfe LS, Kominami E. Ezaki J, et al. Gerontology. 1995;41 Suppl 2:259-69. doi: 10.1159/000213748. Gerontology. 1995. PMID: 8821337

10

Specific delay in the degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis is derived from cellular proteolytic dysfunction rather than structural alteration of subunit c.

Ezaki J, Wolfe LS, Kominami E. Ezaki J, et al. J Neurochem. 1996 Oct;67(4):1677-87. doi: 10.1046/j.1471-4159.1996.67041677.x. J Neurochem. 1996. PMID: 8858953

Save citations to file

Email citations

Send citations to clipboard

Add to Collections

Add to My Bibliography

Create a file for external citation management software

Your saved search

Your RSS Feed

My Custom Filters

Publication date

Text availability

Article attribute

Article type

Article Language

Species

Sex

Age

Other

68 results

Search Page

Filters