Kasa P - Search Results

1

Human amyloid-beta causes changes in the levels of endothelial protein kinase C and its alpha isoform in vitro.

Pakaski M, Balaspiri L, Checler F, Kasa P. Pakaski M, et al. Among authors: kasa p. Neurochem Int. 2002 Dec;41(6):409-14. doi: 10.1016/s0197-0186(02)00026-8. Neurochem Int. 2002. PMID: 12213228

2

C-terminal fragments of amyloid-beta peptide cause cholinergic axonal degeneration by a toxic effect rather than by physical injury in the nondemented human brain.

Kasa P, Papp H, Zombori J, Mayer P, Checler F. Kasa P, et al. Neurochem Res. 2003 Apr;28(3-4):493-8. doi: 10.1023/a:1022813104905. Neurochem Res. 2003. PMID: 12675137

3

Effects of amyloid-beta on cholinergic and acetylcholinesterase-positive cells in cultured basal forebrain neurons of embryonic rat brain.

Kasa P Sr, Papp H, Kasa P Jr, Pakaski M, Balaspiri L. Kasa P Sr, et al. Among authors: kasa p jr. Brain Res. 2004 Feb 13;998(1):73-82. doi: 10.1016/j.brainres.2003.11.021. Brain Res. 2004. PMID: 14725970

4

Imipramine and citalopram facilitate amyloid precursor protein secretion in vitro.

Pákáski M, Bjelik A, Hugyecz M, Kása P, Janka Z, Kálmán J. Pákáski M, et al. Among authors: kasa p. Neurochem Int. 2005 Aug;47(3):190-5. doi: 10.1016/j.neuint.2005.03.004. Neurochem Int. 2005. PMID: 15955598

5

In vitro effects of metrifonate on neuronal amyloid precursor protein processing and protein kinase C level.

Pakaski M, Rakonczay Z, Fakla I, Papp H, Kasa P. Pakaski M, et al. Among authors: kasa p. Brain Res. 2000 Apr 28;863(1-2):266-70. doi: 10.1016/s0006-8993(00)02120-x. Brain Res. 2000. PMID: 10773217

6

Reversible and irreversible acetylcholinesterase inhibitors cause changes in neuronal amyloid precursor protein processing and protein kinase C level in vitro.

Pakaski M, Rakonczay Z, Kasa P. Pakaski M, et al. Among authors: kasa p. Neurochem Int. 2001 Mar;38(3):219-26. doi: 10.1016/s0197-0186(00)00091-7. Neurochem Int. 2001. PMID: 11099780

7

Presenilin-1 and the amyloid precursor protein are transported bidirectionally in the sciatic nerve of adult rat.

Papp H, Pakaski M, Kasa P. Papp H, et al. Among authors: kasa p. Neurochem Int. 2002 Dec;41(6):429-35. doi: 10.1016/s0197-0186(02)00014-1. Neurochem Int. 2002. PMID: 12213230

8

Amyloid-beta1-42 treatment does not have a specific effect on cholinergic neurons in in vitro basal forebrain neuronal cultures of rat.

Papp H, Kása P Jr, Pákáski M, Baláspiri L, Kása P Sr. Papp H, et al. Among authors: kasa p jr, kasa p sr. Acta Biol Hung. 2002;53(3):257-65. doi: 10.1556/ABiol.53.2002.3.2. Acta Biol Hung. 2002. PMID: 12371605

9

Role of acetylcholinesterase inhibitors in the metabolism of amyloid precursor protein.

Pakaski M, Kasa P. Pakaski M, et al. Among authors: kasa p. Curr Drug Targets CNS Neurol Disord. 2003 Jun;2(3):163-71. doi: 10.2174/1568007033482869. Curr Drug Targets CNS Neurol Disord. 2003. PMID: 12769797 Review.

10

Vulnerability of small GABAergic neurons to human beta-amyloid pentapeptide.

Pakaski M, Farkas Z, Kasa P Jr, Forgon M, Papp H, Zarandi M, Penke B, Kasa P Sr. Pakaski M, et al. Among authors: kasa p jr, kasa p sr. Brain Res. 1998 Jun 15;796(1-2):239-46. doi: 10.1016/s0006-8993(98)00358-8. Brain Res. 1998. PMID: 9689474

Save citations to file

Email citations

Send citations to clipboard

Add to Collections

Add to My Bibliography

Create a file for external citation management software

Your saved search

Your RSS Feed

My Custom Filters

Publication date

Text availability

Article attribute

Article type

Article Language

Species

Sex

Age

Other

174 results

Search Page

Filters