Li H - Search Results

1

Anthraquinones sensitize tumor cells to arsenic cytotoxicity in vitro and in vivo via reactive oxygen species-mediated dual regulation of apoptosis.

Yang J, Li H, Chen YY, Wang XJ, Shi GY, Hu QS, Kang XL, Lu Y, Tang XM, Guo QS, Yi J. Yang J, et al. Among authors: li h. Free Radic Biol Med. 2004 Dec 15;37(12):2027-41. doi: 10.1016/j.freeradbiomed.2004.09.016. Free Radic Biol Med. 2004. PMID: 15544921

2

The sensitivity of digestive tract tumor cells to As2O3 is associated with the inherent cellular level of reactive oxygen species.

Gao F, Yi J, Shi GY, Li H, Shi XG, Tang XM. Gao F, et al. Among authors: li h. World J Gastroenterol. 2002 Feb;8(1):36-9. doi: 10.3748/wjg.v8.i1.36. World J Gastroenterol. 2002. PMID: 11833067 Free PMC article.

3

[Ascorbic acid enhances the apoptosis of U937 cells induced by arsenic trioxide in combination with DMNQ and its mechanism].

Gao F, Yi J, Shi G, Li H, Shi X, Wang Z, Tang X. Gao F, et al. Among authors: li h. Zhonghua Xue Ye Xue Za Zhi. 2002 Jan;23(1):9-11. Zhonghua Xue Ye Xue Za Zhi. 2002. PMID: 12015080 Chinese.

4

The Susceptibility of Leukemia Cells to Arsenic Trioxide-induced Apoptosis is Determined by Cellular Reactive Oxygen Species Level.

Gao F, Yi J, Shi GY, Li H, Jin HF, Shi XG, Tang XM. Gao F, et al. Among authors: li h. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2001;33(5):585-589. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2001. PMID: 12040405

5

Apoptosis susceptibility of tumor cells to arsenic trioxide and the inherent cellular level of reactive oxygen species.

Yi J, Gao F, Shi G, Li H, Shi X, Tang X. Yi J, et al. Among authors: li h. Chin Med J (Engl). 2002 Apr;115(4):603-6. Chin Med J (Engl). 2002. PMID: 12133308

6

[Emodin sensitizes HeLa cell to arsenic trioxide induced apoptosis via the reactive oxygen species-mediated signaling pathways].

Yang J, Tang XM, Li H, Shi GY, Zhu P, Jin HF, Yi J. Yang J, et al. Among authors: li h. Shi Yan Sheng Wu Xue Bao. 2003 Dec;36(6):465-75. Shi Yan Sheng Wu Xue Bao. 2003. PMID: 14724938 Chinese.

7

Alteration of subcellular redox equilibrium and the consequent oxidative modification of nuclear factor kappaB are critical for anticancer cytotoxicity by emodin, a reactive oxygen species-producing agent.

Jing Y, Yang J, Wang Y, Li H, Chen Y, Hu Q, Shi G, Tang X, Yi J. Jing Y, et al. Among authors: li h. Free Radic Biol Med. 2006 Jun 15;40(12):2183-97. doi: 10.1016/j.freeradbiomed.2006.02.016. Epub 2006 Mar 24. Free Radic Biol Med. 2006. PMID: 16785032

8

Emodin enhances sensitivity of gallbladder cancer cells to platinum drugs via glutathion depletion and MRP1 downregulation.

Wang W, Sun YP, Huang XZ, He M, Chen YY, Shi GY, Li H, Yi J, Wang J. Wang W, et al. Among authors: li h. Biochem Pharmacol. 2010 Apr 15;79(8):1134-40. doi: 10.1016/j.bcp.2009.12.006. Epub 2009 Dec 11. Biochem Pharmacol. 2010. PMID: 20005210

9

Emodin potentiates the anticancer effect of cisplatin on gallbladder cancer cells through the generation of reactive oxygen species and the inhibition of survivin expression.

Wang W, Sun Y, Li X, Li H, Chen Y, Tian Y, Yi J, Wang J. Wang W, et al. Among authors: li x, li h. Oncol Rep. 2011 Nov;26(5):1143-8. doi: 10.3892/or.2011.1390. Epub 2011 Jul 14. Oncol Rep. 2011. PMID: 21769433

10

Emodin enhances cisplatin-induced cytotoxicity in human bladder cancer cells through ROS elevation and MRP1 downregulation.

Li X, Wang H, Wang J, Chen Y, Yin X, Shi G, Li H, Hu Z, Liang X. Li X, et al. Among authors: li h. BMC Cancer. 2016 Aug 2;16:578. doi: 10.1186/s12885-016-2640-3. BMC Cancer. 2016. PMID: 27485374 Free PMC article.

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