Wang Q - Search Results

1

Cooperation between antioxidants and 1,25-dihydroxyvitamin D3 in induction of leukemia HL60 cell differentiation through the JNK/AP-1/Egr-1 pathway.

Wang Q, Salman H, Danilenko M, Studzinski GP. Wang Q, et al. J Cell Physiol. 2005 Sep;204(3):964-74. doi: 10.1002/jcp.20355. J Cell Physiol. 2005. PMID: 15799027

2

Translational study of vitamin D differentiation therapy of myeloid leukemia: effects of the combination with a p38 MAPK inhibitor and an antioxidant.

Wang Q, Harrison JS, Uskokovic M, Kutner A, Studzinski GP. Wang Q, et al. Leukemia. 2005 Oct;19(10):1812-7. doi: 10.1038/sj.leu.2403916. Leukemia. 2005. PMID: 16107889

3

Jun N-terminal kinase pathway enhances signaling of monocytic differentiation of human leukemia cells induced by 1,25-dihydroxyvitamin D3.

Wang Q, Wang X, Studzinski GP. Wang Q, et al. Among authors: wang x. J Cell Biochem. 2003 Aug 15;89(6):1087-101. doi: 10.1002/jcb.10595. J Cell Biochem. 2003. PMID: 12898508

4

Up-regulation of Egr1 by 1,25-dihydroxyvitamin D3 contributes to increased expression of p35 activator of cyclin-dependent kinase 5 and consequent onset of the terminal phase of HL60 cell differentiation.

Chen F, Wang Q, Wang X, Studzinski GP. Chen F, et al. Among authors: wang x, wang q. Cancer Res. 2004 Aug 1;64(15):5425-33. doi: 10.1158/0008-5472.CAN-04-0806. Cancer Res. 2004. PMID: 15289351

5

The rationale for deltanoids in therapy for myeloid leukemia: role of KSR-MAPK-C/EBP pathway.

Studzinski GP, Wang X, Ji Y, Wang Q, Zhang Y, Kutner A, Harrison JS. Studzinski GP, et al. Among authors: wang x, wang q. J Steroid Biochem Mol Biol. 2005 Oct;97(1-2):47-55. doi: 10.1016/j.jsbmb.2005.06.010. Epub 2005 Jul 25. J Steroid Biochem Mol Biol. 2005. PMID: 16046262 Free PMC article.

6

Cyclin-dependent kinase inhibitor p27 as a mediator of the G1-S phase block induced by 1,25-dihydroxyvitamin D3 in HL60 cells.

Wang QM, Jones JB, Studzinski GP. Wang QM, et al. Cancer Res. 1996 Jan 15;56(2):264-7. Cancer Res. 1996. PMID: 8542578

7

Increased stringency of the 1,25-dihydroxyvitamin D3-induced G1 to S phase block in polyploid HL60 cells.

Zhang F, Rathod B, Jones JB, Wang QM, Bernhard E, Godyn JJ, Studzinski GP. Zhang F, et al. Among authors: wang qm. J Cell Physiol. 1996 Jul;168(1):18-25. doi: 10.1002/(SICI)1097-4652(199607)168:1<18::AID-JCP3>3.0.CO;2-B. J Cell Physiol. 1996. PMID: 8647913

8

Uncoupling of cell cycle arrest from the expression of monocytic differentiation markers in HL60 cell variants.

Studzinski GP, Rathod B, Wang QM, Rao J, Zhang F. Studzinski GP, et al. Among authors: wang qm. Exp Cell Res. 1997 May 1;232(2):376-87. doi: 10.1006/excr.1997.3484. Exp Cell Res. 1997. PMID: 9168815

9

Cyclin-dependent kinase 6 is the principal target of p27/Kip1 regulation of the G1-phase traverse in 1,25-dihydroxyvitamin D3-treated HL60 cells.

Wang QM, Luo X, Studzinski GP. Wang QM, et al. Cancer Res. 1997 Jul 15;57(14):2851-5. Cancer Res. 1997. PMID: 9230188

10

Lowering of p27Kip1 levels by its antisense or by development of resistance to 1,25-dihydroxyvitamin D3 reverses the G1 block but not differentiation of HL60 cells.

Wang QM, Chen F, Luo X, Moore DC, Flanagan M, Studzinski GP. Wang QM, et al. Leukemia. 1998 Aug;12(8):1256-65. doi: 10.1038/sj.leu.2401088. Leukemia. 1998. PMID: 9697881

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