Bea F - Search Results

1

Calcification of advanced atherosclerotic lesions in the innominate arteries of ApoE-deficient mice: potential role of chondrocyte-like cells.

Rattazzi M, Bennett BJ, Bea F, Kirk EA, Ricks JL, Speer M, Schwartz SM, Giachelli CM, Rosenfeld ME. Rattazzi M, et al. Among authors: bea f. Arterioscler Thromb Vasc Biol. 2005 Jul;25(7):1420-5. doi: 10.1161/01.ATV.0000166600.58468.1b. Epub 2005 Apr 21. Arterioscler Thromb Vasc Biol. 2005. PMID: 15845913

2

Simvastatin promotes atherosclerotic plaque stability in apoE-deficient mice independently of lipid lowering.

Bea F, Blessing E, Bennett B, Levitz M, Wallace EP, Rosenfeld ME. Bea F, et al. Arterioscler Thromb Vasc Biol. 2002 Nov 1;22(11):1832-7. doi: 10.1161/01.atv.0000036081.01231.16. Arterioscler Thromb Vasc Biol. 2002. PMID: 12426212

3

Simvastatin inhibits expression of tissue factor in advanced atherosclerotic lesions of apolipoprotein E deficient mice independently of lipid lowering: potential role of simvastatin-mediated inhibition of Egr-1 expression and activation.

Bea F, Blessing E, Shelley MI, Shultz JM, Rosenfeld ME. Bea F, et al. Atherosclerosis. 2003 Apr;167(2):187-94. doi: 10.1016/s0021-9150(02)00387-8. Atherosclerosis. 2003. PMID: 12818400

4

Chronic inhibition of cyclooxygenase-2 does not alter plaque composition in a mouse model of advanced unstable atherosclerosis.

Bea F, Blessing E, Bennett BJ, Kuo CC, Campbell LA, Kreuzer J, Rosenfeld ME. Bea F, et al. Cardiovasc Res. 2003 Oct 15;60(1):198-204. doi: 10.1016/s0008-6363(03)00464-4. Cardiovasc Res. 2003. PMID: 14522423

5

Lesion progression and plaque composition are not altered in older apoE-/- mice lacking tumor necrosis factor-alpha receptor p55.

Blessing E, Bea F, Kuo CC, Campbell LA, Chesebro B, Rosenfeld ME. Blessing E, et al. Among authors: bea f. Atherosclerosis. 2004 Oct;176(2):227-32. doi: 10.1016/j.atherosclerosis.2004.05.033. Atherosclerosis. 2004. PMID: 15380444

6

Melagatran reduces advanced atherosclerotic lesion size and may promote plaque stability in apolipoprotein E-deficient mice.

Bea F, Kreuzer J, Preusch M, Schaab S, Isermann B, Rosenfeld ME, Katus H, Blessing E. Bea F, et al. Arterioscler Thromb Vasc Biol. 2006 Dec;26(12):2787-92. doi: 10.1161/01.ATV.0000246797.05781.ad. Epub 2006 Sep 21. Arterioscler Thromb Vasc Biol. 2006. PMID: 16990551

7

Critical role of macrophages in glucocorticoid driven vascular calcification in a mouse-model of atherosclerosis.

Preusch MR, Rattazzi M, Albrecht C, Merle U, Tuckermann J, Schütz G, Blessing E, Zoppellaro G, Pauletto P, Krempien R, Rosenfeld ME, Katus HA, Bea F. Preusch MR, et al. Among authors: bea f. Arterioscler Thromb Vasc Biol. 2008 Dec;28(12):2158-64. doi: 10.1161/ATVBAHA.108.174128. Epub 2008 Sep 11. Arterioscler Thromb Vasc Biol. 2008. PMID: 18787189

8

Dabigatran etexilate retards the initiation and progression of atherosclerotic lesions and inhibits the expression of oncostatin M in apolipoprotein E-deficient mice.

Preusch MR, Ieronimakis N, Wijelath ES, Cabbage S, Ricks J, Bea F, Reyes M, van Ryn J, Rosenfeld ME. Preusch MR, et al. Among authors: bea f. Drug Des Devel Ther. 2015 Sep 10;9:5203-11. doi: 10.2147/DDDT.S86969. eCollection 2015. Drug Des Devel Ther. 2015. PMID: 26392754 Free PMC article.

9

Foam cell formation inhibits growth of Chlamydia pneumoniae but does not attenuate Chlamydia pneumoniae-induced secretion of proinflammatory cytokines.

Blessing E, Kuo CC, Lin TM, Campbell LA, Bea F, Chesebro B, Rosenfeld ME. Blessing E, et al. Among authors: bea f. Circulation. 2002 Apr 23;105(16):1976-82. doi: 10.1161/01.cir.0000015062.41860.5b. Circulation. 2002. PMID: 11997286

10

Induction of glutathione synthesis in macrophages by oxidized low-density lipoproteins is mediated by consensus antioxidant response elements.

Bea F, Hudson FN, Chait A, Kavanagh TJ, Rosenfeld ME. Bea F, et al. Circ Res. 2003 Mar 7;92(4):386-93. doi: 10.1161/01.RES.0000059561.65545.16. Epub 2003 Feb 6. Circ Res. 2003. PMID: 12600891

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