Halazonetis TD - Search Results

1

Human LATS1 is a mitotic exit network kinase.

Bothos J, Tuttle RL, Ottey M, Luca FC, Halazonetis TD. Bothos J, et al. Among authors: halazonetis td. Cancer Res. 2005 Aug 1;65(15):6568-75. doi: 10.1158/0008-5472.CAN-05-0862. Cancer Res. 2005. PMID: 16061636

2

Defective in mitotic arrest 1/ring finger 8 is a checkpoint protein that antagonizes the human mitotic exit network.

Tuttle RL, Bothos J, Summers MK, Luca FC, Halazonetis TD. Tuttle RL, et al. Among authors: halazonetis td. Mol Cancer Res. 2007 Dec;5(12):1304-11. doi: 10.1158/1541-7786.MCR-07-0388. Mol Cancer Res. 2007. PMID: 18171988

3

The Chfr mitotic checkpoint protein functions with Ubc13-Mms2 to form Lys63-linked polyubiquitin chains.

Bothos J, Summers MK, Venere M, Scolnick DM, Halazonetis TD. Bothos J, et al. Among authors: halazonetis td. Oncogene. 2003 Oct 16;22(46):7101-7. doi: 10.1038/sj.onc.1206831. Oncogene. 2003. PMID: 14562038

4

Inactivating mutations targeting the chfr mitotic checkpoint gene in human lung cancer.

Mariatos G, Bothos J, Zacharatos P, Summers MK, Scolnick DM, Kittas C, Halazonetis TD, Gorgoulis VG. Mariatos G, et al. Among authors: halazonetis td. Cancer Res. 2003 Nov 1;63(21):7185-9. Cancer Res. 2003. PMID: 14612512

5

The CHFR mitotic checkpoint protein delays cell cycle progression by excluding Cyclin B1 from the nucleus.

Summers MK, Bothos J, Halazonetis TD. Summers MK, et al. Among authors: halazonetis td. Oncogene. 2005 Apr 14;24(16):2589-98. doi: 10.1038/sj.onc.1208428. Oncogene. 2005. PMID: 15674323

6

53BP1 and NFBD1/MDC1-Nbs1 function in parallel interacting pathways activating ataxia-telangiectasia mutated (ATM) in response to DNA damage.

Mochan TA, Venere M, DiTullio RA Jr, Halazonetis TD. Mochan TA, et al. Among authors: halazonetis td. Cancer Res. 2003 Dec 15;63(24):8586-91. Cancer Res. 2003. PMID: 14695167

7

Phosphorylation of ATR-interacting protein on Ser239 mediates an interaction with breast-ovarian cancer susceptibility 1 and checkpoint function.

Venere M, Snyder A, Zgheib O, Halazonetis TD. Venere M, et al. Among authors: halazonetis td. Cancer Res. 2007 Jul 1;67(13):6100-5. doi: 10.1158/0008-5472.CAN-07-0369. Cancer Res. 2007. PMID: 17616665

8

Substitutions that compromise the ionizing radiation-induced association of p53 with 14-3-3 proteins also compromise the ability of p53 to induce cell cycle arrest.

Stavridi ES, Chehab NH, Malikzay A, Halazonetis TD. Stavridi ES, et al. Among authors: halazonetis td. Cancer Res. 2001 Oct 1;61(19):7030-3. Cancer Res. 2001. PMID: 11585729

9

An engineered four-stranded coiled coil substitutes for the tetramerization domain of wild-type p53 and alleviates transdominant inhibition by tumor-derived p53 mutants.

Waterman MJ, Waterman JL, Halazonetis TD. Waterman MJ, et al. Among authors: halazonetis td. Cancer Res. 1996 Jan 1;56(1):158-63. Cancer Res. 1996. PMID: 8548757

10

14-3-3 binding sites in the snail protein are essential for snail-mediated transcriptional repression and epithelial-mesenchymal differentiation.

Hou Z, Peng H, White DE, Wang P, Lieberman PM, Halazonetis T, Rauscher FJ 3rd. Hou Z, et al. Cancer Res. 2010 Jun 1;70(11):4385-93. doi: 10.1158/0008-5472.CAN-10-0070. Epub 2010 May 25. Cancer Res. 2010. PMID: 20501852 Free PMC article.

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