Villunger A - Search Results

1

Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis induction.

Erlacher M, Labi V, Manzl C, Böck G, Tzankov A, Häcker G, Michalak E, Strasser A, Villunger A. Erlacher M, et al. Among authors: villunger a. J Exp Med. 2006 Dec 25;203(13):2939-51. doi: 10.1084/jem.20061552. Epub 2006 Dec 18. J Exp Med. 2006. PMID: 17178918 Free PMC article.

2

Modulation of Apo-1/Fas (CD95)-induced programmed cell death in myeloma cells by interferon-alpha 2.

Egle A, Villunger A, Kos M, Böck G, Gruber J, Auer B, Greil R. Egle A, et al. Among authors: villunger a. Eur J Immunol. 1996 Dec;26(12):3119-26. doi: 10.1002/eji.1830261244. Eur J Immunol. 1996. PMID: 8977313

3

Constitutive expression of Fas (Apo-1/CD95) ligand on multiple myeloma cells: a potential mechanism of tumor-induced suppression of immune surveillance.

Villunger A, Egle A, Marschitz I, Kos M, Böck G, Ludwig H, Geley S, Kofler R, Greil R. Villunger A, et al. Blood. 1997 Jul 1;90(1):12-20. Blood. 1997. PMID: 9207432 Free article.

4

Does "death receptor" signaling play a role in tumorigenesis and cancer therapy?

Villunger A, Strasser A. Villunger A, et al. Oncol Res. 1998;10(11-12):541-50. Oncol Res. 1998. PMID: 10367935 Review.

5

The great escape: is immune evasion required for tumor progression?

Villunger A, Strasser A. Villunger A, et al. Nat Med. 1999 Aug;5(8):874-5. doi: 10.1038/11311. Nat Med. 1999. PMID: 10426306 No abstract available.

6

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L).

Huang DC, Hahne M, Schroeter M, Frei K, Fontana A, Villunger A, Newton K, Tschopp J, Strasser A. Huang DC, et al. Among authors: villunger a. Proc Natl Acad Sci U S A. 1999 Dec 21;96(26):14871-6. doi: 10.1073/pnas.96.26.14871. Proc Natl Acad Sci U S A. 1999. PMID: 10611305 Free PMC article.

7

Fas ligand-induced c-Jun kinase activation in lymphoid cells requires extensive receptor aggregation but is independent of DAXX, and Fas-mediated cell death does not involve DAXX, RIP, or RAIDD.

Villunger A, Huang DC, Holler N, Tschopp J, Strasser A. Villunger A, et al. J Immunol. 2000 Aug 1;165(3):1337-43. doi: 10.4049/jimmunol.165.3.1337. J Immunol. 2000. PMID: 10903735

8

Fas ligand, Bcl-2, granulocyte colony-stimulating factor, and p38 mitogen-activated protein kinase: Regulators of distinct cell death and survival pathways in granulocytes.

Villunger A, O'Reilly LA, Holler N, Adams J, Strasser A. Villunger A, et al. J Exp Med. 2000 Sep 4;192(5):647-58. doi: 10.1084/jem.192.5.647. J Exp Med. 2000. PMID: 10974031 Free PMC article.

9

Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikis.

Puthalakath H, Villunger A, O'Reilly LA, Beaumont JG, Coultas L, Cheney RE, Huang DC, Strasser A. Puthalakath H, et al. Among authors: villunger a. Science. 2001 Sep 7;293(5536):1829-32. doi: 10.1126/science.1062257. Science. 2001. PMID: 11546872

10

Essential role for the BH3-only protein Bim but redundant roles for Bax, Bcl-2, and Bcl-w in the control of granulocyte survival.

Villunger A, Scott C, Bouillet P, Strasser A. Villunger A, et al. Blood. 2003 Mar 15;101(6):2393-400. doi: 10.1182/blood-2002-07-2132. Epub 2002 Nov 14. Blood. 2003. PMID: 12433687 Free article.

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