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Biological features (inflammation and neoangiogenesis) and atherosclerotic risk factors in carotid plaques and calcified aortic valve stenosis: two different sites of the same disease?
Mazzone A, Epistolato MC, Gianetti J, Castagnini M, Sassi C, Ceravolo R, Bevilacqua S, Glauber M, Biagini A, Tanganelli P. Mazzone A, et al. Among authors: glauber m. Am J Clin Pathol. 2006 Oct;126(4):494-502. doi: 10.1309/W75NTE5QBC9DXE03. Am J Clin Pathol. 2006. PMID: 16938654
Surgical or Transcatheter Aortic-Valve Replacement.
Miceli A, Lio A, Glauber M. Miceli A, et al. Among authors: glauber m. N Engl J Med. 2017 Jul 13;377(2):196. doi: 10.1056/NEJMc1706234. N Engl J Med. 2017. PMID: 28700841 No abstract available.
Neoangiogenesis, T-lymphocyte infiltration, and heat shock protein-60 are biological hallmarks of an immunomediated inflammatory process in end-stage calcified aortic valve stenosis.
Mazzone A, Epistolato MC, De Caterina R, Storti S, Vittorini S, Sbrana S, Gianetti J, Bevilacqua S, Glauber M, Biagini A, Tanganelli P. Mazzone A, et al. Among authors: glauber m. J Am Coll Cardiol. 2004 May 5;43(9):1670-6. doi: 10.1016/j.jacc.2003.12.041. J Am Coll Cardiol. 2004. PMID: 15120829 Free article.
298 results