Campos EC - Search Results

1

Coronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms.

Rossi MA, Tanowitz HB, Malvestio LM, Celes MR, Campos EC, Blefari V, Prado CM. Rossi MA, et al. Among authors: campos ec. PLoS Negl Trop Dis. 2010 Aug 31;4(8):e674. doi: 10.1371/journal.pntd.0000674. PLoS Negl Trop Dis. 2010. PMID: 20824217 Free PMC article. Review.

2

Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury.

Campos EC, Romano MM, Prado CM, Rossi MA. Campos EC, et al. Int J Exp Pathol. 2008 Oct;89(5):367-81. doi: 10.1111/j.1365-2613.2008.00604.x. Int J Exp Pathol. 2008. PMID: 18808529 Free PMC article.

3

Disruption of sarcolemmal dystrophin and beta-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis.

Celes MR, Torres-Dueñas D, Malvestio LM, Blefari V, Campos EC, Ramos SG, Prado CM, Cunha FQ, Rossi MA. Celes MR, et al. Among authors: campos ec. Lab Invest. 2010 Apr;90(4):531-42. doi: 10.1038/labinvest.2010.3. Epub 2010 Feb 8. Lab Invest. 2010. PMID: 20142806 Free article.

4

Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins.

Celes MR, Torres-Dueñas D, Prado CM, Campos EC, Moreira JE, Cunha FQ, Rossi MA. Celes MR, et al. Among authors: campos ec. Shock. 2010 Mar;33(3):322-31. doi: 10.1097/SHK.0b013e3181b38ef6. Shock. 2010. PMID: 20160610

5

Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy.

Prado CM, Celes MR, Malvestio LM, Campos EC, Silva JS, Jelicks LA, Tanowitz HB, Rossi MA. Prado CM, et al. Among authors: campos ec. Microbes Infect. 2012 Jan;14(1):59-68. doi: 10.1016/j.micinf.2011.08.010. Epub 2011 Aug 31. Microbes Infect. 2012. PMID: 21914488 Free article.

6

Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy.

Campos EC, O'Connell JL, Malvestio LM, Romano MM, Ramos SG, Celes MR, Prado CM, Simões MV, Rossi MA. Campos EC, et al. Eur J Pharmacol. 2011 Nov 30;670(2-3):541-53. doi: 10.1016/j.ejphar.2011.09.021. Epub 2011 Sep 21. Eur J Pharmacol. 2011. PMID: 21946105 Free article.

7

Early detection of doxorubicin myocardial injury by ultrasonic tissue characterization in an experimental animal model.

Romano MM, Pazin-Filho A, O'Connel JL, Simões MV, Schmidt A, Campos ÉC, Rossi M, Maciel BC. Romano MM, et al. Among authors: campos ec. Cardiovasc Ultrasound. 2012 Oct 10;10:40. doi: 10.1186/1476-7120-10-40. Cardiovasc Ultrasound. 2012. PMID: 23046747 Free PMC article.

8

Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis.

Celes MR, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas AC, Spray DC, Tanowitz HB, da Silva JS, Rossi MA. Celes MR, et al. Among authors: campos ec. PLoS One. 2013 Jul 23;8(7):e68809. doi: 10.1371/journal.pone.0068809. Print 2013. PLoS One. 2013. PMID: 23935889 Free PMC article.

9

Activation of Both the Calpain and Ubiquitin-Proteasome Systems Contributes to Septic Cardiomyopathy through Dystrophin Loss/Disruption and mTOR Inhibition.

Freitas AC, Figueiredo MJ, Campos EC, Soave DF, Ramos SG, Tanowitz HB, Celes MR. Freitas AC, et al. Among authors: campos ec. PLoS One. 2016 Nov 23;11(11):e0166839. doi: 10.1371/journal.pone.0166839. eCollection 2016. PLoS One. 2016. PMID: 27880847 Free PMC article.

10

Increased Atrial β-Adrenergic Receptors and GRK-2 Gene Expression Can Play a Fundamental Role in Heart Failure After Repair of Congenital Heart Disease with Cardiopulmonary Bypass.

Oliveira MS, Carmona F, Vicente WVA, Manso PH, Mata KM, Celes MR, Campos EC, Ramos SG. Oliveira MS, et al. Among authors: campos ec. Pediatr Cardiol. 2017 Apr;38(4):734-745. doi: 10.1007/s00246-017-1573-1. Epub 2017 Feb 18. Pediatr Cardiol. 2017. PMID: 28214967

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