Akt is altered in an animal model of Huntington's disease and in patients.
Colin E, Régulier E, Perrin V, Dürr A, Brice A, Aebischer P, Déglon N, Humbert S, Saudou F.
Colin E, et al. Among authors: humbert s.
Eur J Neurosci. 2005 Mar;21(6):1478-88. doi: 10.1111/j.1460-9568.2005.03985.x.
Eur J Neurosci. 2005.
PMID: 15845076
The insulin-like growth factor I (IGF-1)/Akt pathway plays a crucial role in Huntington's disease by phosphorylating the causative protein, polyQ-huntingtin, and abolishing its toxic properties [Humbert et al. (2002)Dev. ...In the present report, we thus aimed to an …
The insulin-like growth factor I (IGF-1)/Akt pathway plays a crucial role in Huntington's disease by phosphorylating the causative pr …