Halaban R - Search Results

1

Integrated analysis of tumor samples sheds light on tumor heterogeneity.

Parisi F, Micsinai M, Strino F, Ariyan S, Narayan D, Bacchiocchi A, Cheng E, Xu F, Li P, Kluger H, Halaban R, Kluger Y. Parisi F, et al. Among authors: halaban r. Yale J Biol Med. 2012 Sep;85(3):347-61. Epub 2012 Sep 25. Yale J Biol Med. 2012. PMID: 23012583 Free PMC article.

2

Identification of p90RSK as the probable CREB-Ser133 kinase in human melanocytes.

Böhm M, Moellmann G, Cheng E, Alvarez-Franco M, Wagner S, Sassone-Corsi P, Halaban R. Böhm M, et al. Among authors: halaban r. Cell Growth Differ. 1995 Mar;6(3):291-302. Cell Growth Differ. 1995. PMID: 7540859

3

Growth regulatory proteins that repress differentiation markers in melanocytes also downregulate the transcription factor microphthalmia.

Halaban R, Böhm M, Dotto P, Moellmann G, Cheng E, Zhang Y. Halaban R, et al. J Invest Dermatol. 1996 Jun;106(6):1266-72. doi: 10.1111/1523-1747.ep12348972. J Invest Dermatol. 1996. PMID: 8752668 Free article.

4

Aberrant retention of tyrosinase in the endoplasmic reticulum mediates accelerated degradation of the enzyme and contributes to the dedifferentiated phenotype of amelanotic melanoma cells.

Halaban R, Cheng E, Zhang Y, Moellmann G, Hanlon D, Michalak M, Setaluri V, Hebert DN. Halaban R, et al. Proc Natl Acad Sci U S A. 1997 Jun 10;94(12):6210-5. doi: 10.1073/pnas.94.12.6210. Proc Natl Acad Sci U S A. 1997. PMID: 9177196 Free PMC article.

5

Release of cell cycle constraints in mouse melanocytes by overexpressed mutant E2F1E132, but not by deletion of p16INK4A or p21WAF1/CIP1.

Halaban R, Cheng E, Zhang Y, Mandigo CE, Miglarese MR. Halaban R, et al. Oncogene. 1998 May 14;16(19):2489-501. doi: 10.1038/sj.onc.1201773. Oncogene. 1998. PMID: 9627115

6

Deregulated E2F transcriptional activity in autonomously growing melanoma cells.

Halaban R, Cheng E, Smicun Y, Germino J. Halaban R, et al. J Exp Med. 2000 Mar 20;191(6):1005-16. doi: 10.1084/jem.191.6.1005. J Exp Med. 2000. PMID: 10727462 Free PMC article.

7

Endoplasmic reticulum retention is a common defect associated with tyrosinase-negative albinism.

Halaban R, Svedine S, Cheng E, Smicun Y, Aron R, Hebert DN. Halaban R, et al. Proc Natl Acad Sci U S A. 2000 May 23;97(11):5889-94. doi: 10.1073/pnas.97.11.5889. Proc Natl Acad Sci U S A. 2000. PMID: 10823941 Free PMC article.

8

Proper folding and endoplasmic reticulum to golgi transport of tyrosinase are induced by its substrates, DOPA and tyrosine.

Halaban R, Cheng E, Svedine S, Aron R, Hebert DN. Halaban R, et al. J Biol Chem. 2001 Apr 13;276(15):11933-8. doi: 10.1074/jbc.M008703200. Epub 2000 Dec 20. J Biol Chem. 2001. PMID: 11124258 Free article.

9

Abnormal acidification of melanoma cells induces tyrosinase retention in the early secretory pathway.

Halaban R, Patton RS, Cheng E, Svedine S, Trombetta ES, Wahl ML, Ariyan S, Hebert DN. Halaban R, et al. J Biol Chem. 2002 Apr 26;277(17):14821-8. doi: 10.1074/jbc.M111497200. Epub 2002 Jan 25. J Biol Chem. 2002. PMID: 11812790 Free article.

10

Coexpression of wild-type tyrosinase enhances maturation of temperature-sensitive tyrosinase mutants.

Halaban R, Cheng E, Hebert DN. Halaban R, et al. J Invest Dermatol. 2002 Aug;119(2):481-8. doi: 10.1046/j.1523-1747.2002.01824.x. J Invest Dermatol. 2002. PMID: 12190874 Free article.

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