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Keratitis-ichthyosis-deafness syndrome-associated Cx26 mutants produce nonfunctional gap junctions but hyperactive hemichannels when co-expressed with wild type Cx43.
García IE, Maripillán J, Jara O, Ceriani R, Palacios-Muñoz A, Ramachandran J, Olivero P, Perez-Acle T, González C, Sáez JC, Contreras JE, Martínez AD. García IE, et al. J Invest Dermatol. 2015 May;135(5):1338-1347. doi: 10.1038/jid.2015.20. Epub 2015 Jan 27. J Invest Dermatol. 2015. PMID: 25625422 Free PMC article.
On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells.
Vargas AA, Cisterna BA, Saavedra-Leiva F, Urrutia C, Cea LA, Vielma AH, Gutierrez-Maldonado SE, Martin AJ, Pareja-Barrueto C, Escalona Y, Schmachtenberg O, Lagos CF, Perez-Acle T, Sáez JC. Vargas AA, et al. Front Physiol. 2017 Feb 9;8:38. doi: 10.3389/fphys.2017.00038. eCollection 2017. Front Physiol. 2017. PMID: 28232803 Free PMC article.
Expression of KID syndromic mutation Cx26S17F produces hyperactive hemichannels in supporting cells of the organ of Corti.
Abbott AC, García IE, Villanelo F, Flores-Muñoz C, Ceriani R, Maripillán J, Novoa-Molina J, Figueroa-Cares C, Pérez-Acle T, Sáez JC, Sánchez HA, Martínez AD. Abbott AC, et al. Among authors: perez acle t. Front Cell Dev Biol. 2023 Jan 9;10:1071202. doi: 10.3389/fcell.2022.1071202. eCollection 2022. Front Cell Dev Biol. 2023. PMID: 36699003 Free PMC article.
54 results