Singh IN - Search Results

1

Phenelzine Protects Brain Mitochondrial Function In Vitro and In Vivo following Traumatic Brain Injury by Scavenging the Reactive Carbonyls 4-Hydroxynonenal and Acrolein Leading to Cortical Histological Neuroprotection.

Cebak JE, Singh IN, Hill RL, Wang JA, Hall ED. Cebak JE, et al. Among authors: singh in. J Neurotrauma. 2017 Apr 1;34(7):1302-1317. doi: 10.1089/neu.2016.4624. Epub 2016 Dec 2. J Neurotrauma. 2017. PMID: 27750484 Free PMC article.

2

Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.

Vaishnav RA, Singh IN, Miller DM, Hall ED. Vaishnav RA, et al. Among authors: singh in. J Neurotrauma. 2010 Jul;27(7):1311-20. doi: 10.1089/neu.2009.1172. J Neurotrauma. 2010. PMID: 20392143 Free PMC article.

3

Mitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals.

Mustafa AG, Singh IN, Wang J, Carrico KM, Hall ED. Mustafa AG, et al. Among authors: singh in. J Neurochem. 2010 Jul;114(1):271-80. doi: 10.1111/j.1471-4159.2010.06749.x. Epub 2010 Apr 16. J Neurochem. 2010. PMID: 20403083 Free PMC article.

4

Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.

Miller DM, Singh IN, Wang JA, Hall ED. Miller DM, et al. Among authors: singh in. Free Radic Biol Med. 2013 Apr;57:1-9. doi: 10.1016/j.freeradbiomed.2012.12.011. Epub 2012 Dec 27. Free Radic Biol Med. 2013. PMID: 23275005 Free PMC article.

5

Phenelzine mitochondrial functional preservation and neuroprotection after traumatic brain injury related to scavenging of the lipid peroxidation-derived aldehyde 4-hydroxy-2-nonenal.

Singh IN, Gilmer LK, Miller DM, Cebak JE, Wang JA, Hall ED. Singh IN, et al. J Cereb Blood Flow Metab. 2013 Apr;33(4):593-9. doi: 10.1038/jcbfm.2012.211. Epub 2013 Jan 16. J Cereb Blood Flow Metab. 2013. PMID: 23321786 Free PMC article.

6

Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.

Miller DM, Singh IN, Wang JA, Hall ED. Miller DM, et al. Among authors: singh in. Exp Neurol. 2015 Feb;264:103-10. doi: 10.1016/j.expneurol.2014.11.008. Epub 2014 Nov 26. Exp Neurol. 2015. PMID: 25432068 Free PMC article.

7

Lipid peroxidation in brain or spinal cord mitochondria after injury.

Hall ED, Wang JA, Bosken JM, Singh IN. Hall ED, et al. Among authors: singh in. J Bioenerg Biomembr. 2016 Apr;48(2):169-74. doi: 10.1007/s10863-015-9600-5. J Bioenerg Biomembr. 2016. PMID: 25595872 Free PMC article. Review.

8

Synaptic Mitochondria Sustain More Damage than Non-Synaptic Mitochondria after Traumatic Brain Injury and Are Protected by Cyclosporine A.

Kulbe JR, Hill RL, Singh IN, Wang JA, Hall ED. Kulbe JR, et al. Among authors: singh in. J Neurotrauma. 2017 Apr 1;34(7):1291-1301. doi: 10.1089/neu.2016.4628. Epub 2016 Oct 13. J Neurotrauma. 2017. PMID: 27596283 Free PMC article.

9

Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury.

Hill RL, Singh IN, Wang JA, Hall ED. Hill RL, et al. Among authors: singh in. Neurochem Int. 2017 Dec;111:45-56. doi: 10.1016/j.neuint.2017.03.015. Epub 2017 Mar 23. Neurochem Int. 2017. PMID: 28342966 Free PMC article.

10

Continuous Infusion of Phenelzine, Cyclosporine A, or Their Combination: Evaluation of Mitochondrial Bioenergetics, Oxidative Damage, and Cytoskeletal Degradation following Severe Controlled Cortical Impact Traumatic Brain Injury in Rats.

Kulbe JR, Singh IN, Wang JA, Cebak JE, Hall ED. Kulbe JR, et al. Among authors: singh in. J Neurotrauma. 2018 Jun 1;35(11):1280-1293. doi: 10.1089/neu.2017.5353. Epub 2018 Mar 27. J Neurotrauma. 2018. PMID: 29336204 Free PMC article.

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