Hay N - Search Results

1

Cell-Autonomous versus Systemic Akt Isoform Deletions Uncovered New Roles for Akt1 and Akt2 in Breast Cancer.

Chen X, Ariss MM, Ramakrishnan G, Nogueira V, Blaha C, Putzbach W, Islam ABMMK, Frolov MV, Hay N. Chen X, et al. Among authors: hay n. Mol Cell. 2020 Oct 1;80(1):87-101.e5. doi: 10.1016/j.molcel.2020.08.017. Epub 2020 Sep 14. Mol Cell. 2020. PMID: 32931746 Free PMC article.

2

Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2.

Peng XD, Xu PZ, Chen ML, Hahn-Windgassen A, Skeen J, Jacobs J, Sundararajan D, Chen WS, Crawford SE, Coleman KG, Hay N. Peng XD, et al. Among authors: hay n. Genes Dev. 2003 Jun 1;17(11):1352-65. doi: 10.1101/gad.1089403. Genes Dev. 2003. PMID: 12782654 Free PMC article.

3

Akt inhibits apoptosis downstream of BID cleavage via a glucose-dependent mechanism involving mitochondrial hexokinases.

Majewski N, Nogueira V, Robey RB, Hay N. Majewski N, et al. Among authors: hay n. Mol Cell Biol. 2004 Jan;24(2):730-40. doi: 10.1128/MCB.24.2.730-740.2004. Mol Cell Biol. 2004. PMID: 14701745 Free PMC article.

4

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak.

Majewski N, Nogueira V, Bhaskar P, Coy PE, Skeen JE, Gottlob K, Chandel NS, Thompson CB, Robey RB, Hay N. Majewski N, et al. Among authors: hay n. Mol Cell. 2004 Dec 3;16(5):819-30. doi: 10.1016/j.molcel.2004.11.014. Mol Cell. 2004. PMID: 15574336 Free article.

5

Akt activates the mammalian target of rapamycin by regulating cellular ATP level and AMPK activity.

Hahn-Windgassen A, Nogueira V, Chen CC, Skeen JE, Sonenberg N, Hay N. Hahn-Windgassen A, et al. Among authors: hay n. J Biol Chem. 2005 Sep 16;280(37):32081-9. doi: 10.1074/jbc.M502876200. Epub 2005 Jul 15. J Biol Chem. 2005. PMID: 16027121 Free article.

6

The Akt-mTOR tango and its relevance to cancer.

Hay N. Hay N. Cancer Cell. 2005 Sep;8(3):179-83. doi: 10.1016/j.ccr.2005.08.008. Cancer Cell. 2005. PMID: 16169463 Free article. Review.

7

The deficiency of Akt1 is sufficient to suppress tumor development in Pten+/- mice.

Chen ML, Xu PZ, Peng XD, Chen WS, Guzman G, Yang X, Di Cristofano A, Pandolfi PP, Hay N. Chen ML, et al. Among authors: hay n. Genes Dev. 2006 Jun 15;20(12):1569-74. doi: 10.1101/gad.1395006. Genes Dev. 2006. PMID: 16778075 Free PMC article.

8

Akt deficiency impairs normal cell proliferation and suppresses oncogenesis in a p53-independent and mTORC1-dependent manner.

Skeen JE, Bhaskar PT, Chen CC, Chen WS, Peng XD, Nogueira V, Hahn-Windgassen A, Kiyokawa H, Hay N. Skeen JE, et al. Among authors: hay n. Cancer Cell. 2006 Oct;10(4):269-80. doi: 10.1016/j.ccr.2006.08.022. Cancer Cell. 2006. PMID: 17045205 Free article.

9

The two TORCs and Akt.

Bhaskar PT, Hay N. Bhaskar PT, et al. Among authors: hay n. Dev Cell. 2007 Apr;12(4):487-502. doi: 10.1016/j.devcel.2007.03.020. Dev Cell. 2007. PMID: 17419990 Free article. Review.

10

Akt determines replicative senescence and oxidative or oncogenic premature senescence and sensitizes cells to oxidative apoptosis.

Nogueira V, Park Y, Chen CC, Xu PZ, Chen ML, Tonic I, Unterman T, Hay N. Nogueira V, et al. Among authors: hay n. Cancer Cell. 2008 Dec 9;14(6):458-70. doi: 10.1016/j.ccr.2008.11.003. Cancer Cell. 2008. PMID: 19061837 Free PMC article.

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