Maeda S - Search Results

1

Establishment of KEIOi005-A iPSC line from urine-derived cells (UDCs) of a mild Alzheimer's disease (AD) donor with multiple risk SNPs for sporadic Alzheimer's disease (sAD).

Supakul S, Leventoux N, Tabuchi H, Mimura M, Ito D, Maeda S, Okano H. Supakul S, et al. Among authors: maeda s. Stem Cell Res. 2022 Jul;62:102802. doi: 10.1016/j.scr.2022.102802. Epub 2022 May 5. Stem Cell Res. 2022. PMID: 35537244 Free article.

2

Pathological Progression Induced by the Frontotemporal Dementia-Associated R406W Tau Mutation in Patient-Derived iPSCs.

Nakamura M, Shiozawa S, Tsuboi D, Amano M, Watanabe H, Maeda S, Kimura T, Yoshimatsu S, Kisa F, Karch CM, Miyasaka T, Takashima A, Sahara N, Hisanaga SI, Ikeuchi T, Kaibuchi K, Okano H. Nakamura M, et al. Among authors: maeda s. Stem Cell Reports. 2019 Oct 8;13(4):684-699. doi: 10.1016/j.stemcr.2019.08.011. Epub 2019 Sep 19. Stem Cell Reports. 2019. PMID: 31543469 Free PMC article.

3

Utilization of Human Induced Pluripotent Stem Cells-Derived In vitro Models for the Future Study of Sex Differences in Alzheimer's Disease.

Supakul S, Okano H, Maeda S. Supakul S, et al. Among authors: maeda s. Front Aging Neurosci. 2021 Nov 4;13:768948. doi: 10.3389/fnagi.2021.768948. eCollection 2021. Front Aging Neurosci. 2021. PMID: 34803659 Free PMC article.

4

Integrated Collection of Stem Cell Bank Data, a Data Portal for Standardized Stem Cell Information.

Chen Y, Sakurai K, Maeda S, Masui T, Okano H, Dewender J, Seltmann S, Kurtz A, Masuya H, Nakamura Y, Sheldon M, Schneider J, Stacey GN, Panina Y, Fujibuchi W. Chen Y, et al. Among authors: maeda s. Stem Cell Reports. 2021 Apr 13;16(4):997-1005. doi: 10.1016/j.stemcr.2021.02.014. Epub 2021 Mar 18. Stem Cell Reports. 2021. PMID: 33740463 Free PMC article.

5

Tau Oligomers.

Maeda S, Takashima A. Maeda S, et al. Adv Exp Med Biol. 2019;1184:373-380. doi: 10.1007/978-981-32-9358-8_27. Adv Exp Med Biol. 2019. PMID: 32096050 Review.

6

Methylene Blue Inhibits Formation of Tau Fibrils but not of Granular Tau Oligomers: A Plausible Key to Understanding Failure of a Clinical Trial for Alzheimer's Disease.

Soeda Y, Saito M, Maeda S, Ishida K, Nakamura A, Kojima S, Takashima A. Soeda Y, et al. Among authors: maeda s. J Alzheimers Dis. 2019;68(4):1677-1686. doi: 10.3233/JAD-181001. J Alzheimers Dis. 2019. PMID: 30909223

7

Frontotemporal dementia with Parkinsonism linked to chromosome-17 mutations enhance tau oligomer formation.

Maeda S, Sato Y, Takashima A. Maeda S, et al. Neurobiol Aging. 2018 Sep;69:26-32. doi: 10.1016/j.neurobiolaging.2018.04.014. Epub 2018 May 7. Neurobiol Aging. 2018. PMID: 29852407

8

Tau oligomerization: a role for tau aggregation intermediates linked to neurodegeneration.

Sahara N, Maeda S, Takashima A. Sahara N, et al. Among authors: maeda s. Curr Alzheimer Res. 2008 Dec;5(6):591-8. doi: 10.2174/156720508786898442. Curr Alzheimer Res. 2008. PMID: 19075586 Review.

9

Increased levels of granular tau oligomers: an early sign of brain aging and Alzheimer's disease.

Maeda S, Sahara N, Saito Y, Murayama S, Ikai A, Takashima A. Maeda S, et al. Neurosci Res. 2006 Mar;54(3):197-201. doi: 10.1016/j.neures.2005.11.009. Epub 2006 Jan 6. Neurosci Res. 2006. PMID: 16406150

10

Tau Phosphorylation-Much More than a Biomarker.

Maeda S, Mucke L. Maeda S, et al. Neuron. 2016 Oct 19;92(2):265-267. doi: 10.1016/j.neuron.2016.10.011. Neuron. 2016. PMID: 27764656 Free article.

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