Ide T - Search Results

1

Ethoxyquin is a Competent Radical-Trapping Antioxidant for Preventing Ferroptosis in Doxorubicin Cardiotoxicity.

Tadokoro T, Ikeda M, Abe K, Ide T, Miyamoto HD, Furusawa S, Ishimaru K, Watanabe M, Ishikita A, Matsushima S, Koumura T, Yamada KI, Imai H, Tsutsui H. Tadokoro T, et al. Among authors: ide t. J Cardiovasc Pharmacol. 2022 Nov 1;80(5):690-699. doi: 10.1097/FJC.0000000000001328. J Cardiovasc Pharmacol. 2022. PMID: 35881422

2

Amiodarone protects cardiac myocytes against oxidative injury by its free radical scavenging action.

Ide T, Tsutsui H, Kinugawa S, Utsumi H, Takeshita A. Ide T, et al. Circulation. 1999 Aug 17;100(7):690-2. doi: 10.1161/01.cir.100.7.690. Circulation. 1999. PMID: 10449688

3

Mitochondrial electron transport complex I is a potential source of oxygen free radicals in the failing myocardium.

Ide T, Tsutsui H, Kinugawa S, Utsumi H, Kang D, Hattori N, Uchida K, Arimura Ki, Egashira K, Takeshita A. Ide T, et al. Circ Res. 1999 Aug 20;85(4):357-63. doi: 10.1161/01.res.85.4.357. Circ Res. 1999. PMID: 10455064

4

Positive inotropic effect of insulin-like growth factor-1 on normal and failing cardiac myocytes.

Kinugawa S, Tsutsui H, Ide T, Nakamura R, Arimura K, Egashira K, Takeshita A. Kinugawa S, et al. Among authors: ide t. Cardiovasc Res. 1999 Jul;43(1):157-64. doi: 10.1016/s0008-6363(99)00058-9. Cardiovasc Res. 1999. PMID: 10536700

5

Role of Ca2+ availability to myofilaments and their sensitivity to Ca2+ in myocyte contractile dysfunction in heart failure.

Kinugawa S, Tsutsui H, Satoh S, Takahashi M, Ide T, IgarashiSaito K, Arimura K, Egashira K, Takeshita A. Kinugawa S, et al. Among authors: ide t. Cardiovasc Res. 1999 Nov;44(2):398-406. doi: 10.1016/s0008-6363(99)00205-9. Cardiovasc Res. 1999. PMID: 10690316

6

Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: role of oxidative stress.

Kinugawa S, Tsutsui H, Hayashidani S, Ide T, Suematsu N, Satoh S, Utsumi H, Takeshita A. Kinugawa S, et al. Among authors: ide t. Circ Res. 2000 Sep 1;87(5):392-8. doi: 10.1161/01.res.87.5.392. Circ Res. 2000. PMID: 10969037

7

Greater susceptibility of failing cardiac myocytes to oxygen free radical-mediated injury.

Tsutsui H, Ide T, Hayashidani S, Suematsu N, Utsumi H, Nakamura R, Egashira K, Takeshita A. Tsutsui H, et al. Among authors: ide t. Cardiovasc Res. 2001 Jan;49(1):103-9. doi: 10.1016/s0008-6363(00)00197-8. Cardiovasc Res. 2001. PMID: 11121801

8

Increased inactivation of nitric oxide is involved in coronary endothelial dysfunction in heart failure.

Arimura K, Egashira K, Nakamura R, Ide T, Tsutsui H, Shimokawa H, Takeshita A. Arimura K, et al. Among authors: ide t. Am J Physiol Heart Circ Physiol. 2001 Jan;280(1):H68-75. doi: 10.1152/ajpheart.2001.280.1.H68. Am J Physiol Heart Circ Physiol. 2001. PMID: 11123219 Free article.

9

Positive inotropic effects of calcium sensitizers on normal and failing cardiac myocytes.

Tsutsui H, Kinugawa S, Ide T, Hayashidani S, Suematsu N, Satoh S, Nakamura R, Egashira K, Takeshita A. Tsutsui H, et al. Among authors: ide t. J Cardiovasc Pharmacol. 2001 Jan;37(1):16-24. doi: 10.1097/00005344-200101000-00003. J Cardiovasc Pharmacol. 2001. PMID: 11152370

10

Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction.

Ide T, Tsutsui H, Hayashidani S, Kang D, Suematsu N, Nakamura K, Utsumi H, Hamasaki N, Takeshita A. Ide T, et al. Circ Res. 2001 Mar 16;88(5):529-35. doi: 10.1161/01.res.88.5.529. Circ Res. 2001. PMID: 11249877

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