Park KM - Search Results

1

Kidney ischemia/reperfusion injury causes cholangiocytes primary cilia disruption and abnormal bile secretion.

Han YK, Lim HJ, Jang G, Jang SY, Park KM. Han YK, et al. Among authors: park km. Biochim Biophys Acta Mol Basis Dis. 2024 Aug;1870(6):167225. doi: 10.1016/j.bbadis.2024.167225. Epub 2024 May 15. Biochim Biophys Acta Mol Basis Dis. 2024. PMID: 38749218

2

Cisplatin induces lung cell cilia disruption and lung damage via oxidative stress.

Han YK, Kim JS, Jang G, Park KM. Han YK, et al. Among authors: park km. Free Radic Biol Med. 2021 Dec;177:270-277. doi: 10.1016/j.freeradbiomed.2021.10.032. Epub 2021 Oct 26. Free Radic Biol Med. 2021. PMID: 34710564 Free article.

3

Oxidative stress following acute kidney injury causes disruption of lung cell cilia and their release into the bronchoaveolar lavage fluid and lung injury, which are exacerbated by Idh2 deletion.

Han YK, Kim JS, Lee GB, Lim JH, Park KM. Han YK, et al. Among authors: park km. Redox Biol. 2021 Oct;46:102077. doi: 10.1016/j.redox.2021.102077. Epub 2021 Jul 21. Redox Biol. 2021. PMID: 34315110 Free PMC article.

4

Short-term control of diet affects cisplatin-induced acute kidney injury through modulation of mitochondrial dynamics and mitochondrial GSH.

Kim JS, Han YK, Kong MJ, Park KM. Kim JS, et al. Among authors: park km. Physiol Rep. 2022 Jun;10(12):e15348. doi: 10.14814/phy2.15348. Physiol Rep. 2022. PMID: 35748040 Free PMC article.

5

Hepatic ischemia/reperfusion injury disrupts the homeostasis of kidney primary cilia via oxidative stress.

Han SJ, Jang HS, Seu SY, Cho HJ, Hwang YJ, Kim JI, Park KM. Han SJ, et al. Among authors: park km. Biochim Biophys Acta Mol Basis Dis. 2017 Jul;1863(7):1817-1828. doi: 10.1016/j.bbadis.2017.05.004. Epub 2017 May 8. Biochim Biophys Acta Mol Basis Dis. 2017. PMID: 28495528 Free article.

6

Inhibition of HDACs (Histone Deacetylases) Ameliorates High-Fat Diet-Induced Hypertension Through Restoration of the MsrA (Methionine Sulfoxide Reductase A)/Hydrogen Sulfide Axis.

Jung JK, Yoon GE, Jang G, Park KM, Kim I, Kim JI. Jung JK, et al. Among authors: park km. Hypertension. 2021 Sep;78(4):1103-1115. doi: 10.1161/HYPERTENSIONAHA.121.17149. Epub 2021 Aug 15. Hypertension. 2021. PMID: 34397279

7

Ablation of C/EBP homologous protein attenuates renal fibrosis after ureteral obstruction by reducing autophagy and microtubule disruption.

Noh MR, Woo CH, Park MJ, In Kim J, Park KM. Noh MR, et al. Among authors: park mj, park km. Biochim Biophys Acta Mol Basis Dis. 2018 May;1864(5 Pt A):1634-1641. doi: 10.1016/j.bbadis.2018.02.001. Epub 2018 Feb 7. Biochim Biophys Acta Mol Basis Dis. 2018. PMID: 29425932 Free article.

8

Activation of ERK accelerates repair of renal tubular epithelial cells, whereas it inhibits progression of fibrosis following ischemia/reperfusion injury.

Jang HS, Han SJ, Kim JI, Lee S, Lipschutz JH, Park KM. Jang HS, et al. Among authors: park km. Biochim Biophys Acta. 2013 Dec;1832(12):1998-2008. doi: 10.1016/j.bbadis.2013.07.001. Epub 2013 Jul 10. Biochim Biophys Acta. 2013. PMID: 23851027 Free article.

9

C/EBP homologous protein (CHOP) gene deficiency attenuates renal ischemia/reperfusion injury in mice.

Noh MR, Kim JI, Han SJ, Lee TJ, Park KM. Noh MR, et al. Among authors: park km. Biochim Biophys Acta. 2015 Sep;1852(9):1895-901. doi: 10.1016/j.bbadis.2015.06.004. Epub 2015 Jun 11. Biochim Biophys Acta. 2015. PMID: 26071644 Free article.

10

Involvement of hydrogen sulfide and homocysteine transsulfuration pathway in the progression of kidney fibrosis after ureteral obstruction.

Jung KJ, Jang HS, Kim JI, Han SJ, Park JW, Park KM. Jung KJ, et al. Among authors: park jw, park km. Biochim Biophys Acta. 2013 Dec;1832(12):1989-97. doi: 10.1016/j.bbadis.2013.06.015. Epub 2013 Jul 9. Biochim Biophys Acta. 2013. PMID: 23846016 Free article.

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