Linch DC - Search Results

1

Analysis of mutations in the GM-CSF receptor alpha coding sequence in patients with acute myeloid leukaemia and haematologically normal individuals by RT-PCR-SSCP.

Wagner HM, Gale RE, Freeburn RW, Devereux S, Linch DC. Wagner HM, et al. Among authors: linch dc. Leukemia. 1994 Sep;8(9):1527-32. Leukemia. 1994. PMID: 7522290

2

Prognostic implications of the presence of FLT3 mutations in patients with acute myeloid leukemia.

Kottaridis PD, Gale RE, Linch DC. Kottaridis PD, et al. Among authors: linch dc. Leuk Lymphoma. 2003 Jun;44(6):905-13. doi: 10.1080/1042819031000067503. Leuk Lymphoma. 2003. PMID: 12854887 Review.

3

ASXL1 mutations are infrequent in young patients with primary acute myeloid leukemia and their detection has a limited role in therapeutic risk stratification.

El-Sharkawi D, Ali A, Evans CM, Hills RK, Burnett AK, Linch DC, Gale RE. El-Sharkawi D, et al. Among authors: linch dc. Leuk Lymphoma. 2014 Jun;55(6):1326-31. doi: 10.3109/10428194.2013.833332. Epub 2013 Oct 9. Leuk Lymphoma. 2014. PMID: 23952244

4

Additional impact of mutational genotype on prognostic determination in resistant and relapsed acute myeloid leukaemia.

Linch DC, Hills RK, Gilkes A, Burnett AK, Russell N, Gale RE. Linch DC, et al. Leuk Res. 2021 Sep;108:106553. doi: 10.1016/j.leukres.2021.106553. Epub 2021 Mar 2. Leuk Res. 2021. PMID: 33706968

5

Frequency of clonal remission in acute myeloid leukaemia.

Gale RE, Wheadon H, Goldstone AH, Burnett AK, Linch DC. Gale RE, et al. Among authors: linch dc. Lancet. 1993 Jan 16;341(8838):138-42. doi: 10.1016/0140-6736(93)90004-z. Lancet. 1993. PMID: 8093744

6

Investigation of methylation at Hha I sites using the hypervariable probe M27 beta allows improved clonal analysis in myeloid leukaemia and demonstrates differences in methylation between leukaemic and remission samples.

Gale RE, Linch DC. Gale RE, et al. Among authors: linch dc. Leukemia. 1994 Jan;8(1):190-4. Leukemia. 1994. PMID: 8289487

7

The beta subunit common to the GM-CSF, IL-3 and IL-5 receptors is highly polymorphic but pathogenic point mutations in patients with acute myeloid leukaemia (AML) are rare.

Freeburn RW, Gale RE, Wagner HM, Linch DC. Freeburn RW, et al. Among authors: linch dc. Leukemia. 1996 Jan;10(1):123-9. Leukemia. 1996. PMID: 8558916

8

Demonstration of developing myelodysplasia/acute myeloid leukaemia in haematologically normal patients after high-dose chemotherapy and autologous bone marrow transplantation using X-chromosome inactivation patterns.

Gale RE, Bunch C, Moir DJ, Patterson KG, Goldstone AH, Linch DC. Gale RE, et al. Among authors: linch dc. Br J Haematol. 1996 Apr;93(1):53-8. doi: 10.1046/j.1365-2141.1996.4751014.x. Br J Haematol. 1996. PMID: 8611475

9

Variable expression of p16 protein in patients with acute myeloid leukemia without gross rearrangements at the DNA level.

Jamal R, Thomas NS, Gale RE, Linch DC. Jamal R, et al. Among authors: linch dc. Leukemia. 1996 Apr;10(4):629-36. Leukemia. 1996. PMID: 8618439

10

Expression of two alternatively spliced forms of the 5' untranslated region of the GM-CSF receptor alpha chain mRNA.

Chopra R, Kendall G, Gale RE, Thomas NS, Linch DC. Chopra R, et al. Among authors: linch dc. Exp Hematol. 1996 May;24(6):755-62. Exp Hematol. 1996. PMID: 8635532

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