Lowenson JD - Search Results

1

D-aspartate content of erythrocyte membrane proteins is decreased in uremia: implications for the repair of damaged proteins.

Perna AF, D'Aniello A, Lowenson JD, Clarke S, De Santo NG, Ingrosso D. Perna AF, et al. Among authors: lowenson jd. J Am Soc Nephrol. 1997 Jan;8(1):95-104. doi: 10.1681/ASN.V8195. J Am Soc Nephrol. 1997. PMID: 9013453

2

Does the chemical instability of aspartyl and asparaginyl residues in proteins contribute to erythrocyte aging? The role of protein carboxyl methylation reactions.

Lowenson J, Clarke S. Lowenson J, et al. Blood Cells. 1988;14(1):103-18. Blood Cells. 1988. PMID: 3052632 Review.

3

Recognition of D-aspartyl residues in polypeptides by the erythrocyte L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis.

Lowenson JD, Clarke S. Lowenson JD, et al. J Biol Chem. 1992 Mar 25;267(9):5985-95. J Biol Chem. 1992. PMID: 1556110 Free article.

4

Spontaneous degradation and enzymatic repair of aspartyl and asparaginyl residues in aging red cell proteins analyzed by computer simulation.

Lowenson JD, Clarke S. Lowenson JD, et al. Gerontology. 1991;37(1-3):128-51. doi: 10.1159/000213255. Gerontology. 1991. PMID: 1829049

5

Structural elements affecting the recognition of L-isoaspartyl residues by the L-isoaspartyl/D-aspartyl protein methyltransferase. Implications for the repair hypothesis.

Lowenson JD, Clarke S. Lowenson JD, et al. J Biol Chem. 1991 Oct 15;266(29):19396-406. J Biol Chem. 1991. PMID: 1833402 Free article.

6

Deficiency of a protein-repair enzyme results in the accumulation of altered proteins, retardation of growth, and fatal seizures in mice.

Kim E, Lowenson JD, MacLaren DC, Clarke S, Young SG. Kim E, et al. Among authors: lowenson jd. Proc Natl Acad Sci U S A. 1997 Jun 10;94(12):6132-7. doi: 10.1073/pnas.94.12.6132. Proc Natl Acad Sci U S A. 1997. PMID: 9177182 Free PMC article.

7

Protein repair L-isoaspartyl methyltransferase in plants. Phylogenetic distribution and the accumulation of substrate proteins in aged barley seeds.

Mudgett MB, Lowenson JD, Clarke S. Mudgett MB, et al. Among authors: lowenson jd. Plant Physiol. 1997 Dec;115(4):1481-9. doi: 10.1104/pp.115.4.1481. Plant Physiol. 1997. PMID: 9414558 Free PMC article.

8

Phenotypic analysis of seizure-prone mice lacking L-isoaspartate (D-aspartate) O-methyltransferase.

Kim E, Lowenson JD, Clarke S, Young SG. Kim E, et al. Among authors: lowenson jd. J Biol Chem. 1999 Jul 16;274(29):20671-8. doi: 10.1074/jbc.274.29.20671. J Biol Chem. 1999. PMID: 10400700 Free article.

9

Limited accumulation of damaged proteins in l-isoaspartyl (D-aspartyl) O-methyltransferase-deficient mice.

Lowenson JD, Kim E, Young SG, Clarke S. Lowenson JD, et al. J Biol Chem. 2001 Jun 8;276(23):20695-702. doi: 10.1074/jbc.M100987200. Epub 2001 Mar 7. J Biol Chem. 2001. PMID: 11279164 Free article.

10

Identification of isoaspartyl-containing sequences in peptides and proteins that are usually poor substrates for the class II protein carboxyl methyltransferase.

Lowenson JD, Clarke S. Lowenson JD, et al. J Biol Chem. 1990 Feb 25;265(6):3106-10. J Biol Chem. 1990. PMID: 2303443 Free article.

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