Smits R - Search Results

1

Loss of Apc and the entire chromosome 18 but absence of mutations at the Ras and Tp53 genes in intestinal tumors from Apc1638N, a mouse model for Apc-driven carcinogenesis.

Smits R, Kartheuser A, Jagmohan-Changur S, Leblanc V, Breukel C, de Vries A, van Kranen H, van Krieken JH, Williamson S, Edelmann W, Kucherlapati R, KhanPM, Fodde R. Smits R, et al. Carcinogenesis. 1997 Feb;18(2):321-7. doi: 10.1093/carcin/18.2.321. Carcinogenesis. 1997. PMID: 9054624

2

A new conditional Apc-mutant mouse model for colorectal cancer.

Robanus-Maandag EC, Koelink PJ, Breukel C, Salvatori DC, Jagmohan-Changur SC, Bosch CA, Verspaget HW, Devilee P, Fodde R, Smits R. Robanus-Maandag EC, et al. Among authors: smits r. Carcinogenesis. 2010 May;31(5):946-52. doi: 10.1093/carcin/bgq046. Epub 2010 Feb 22. Carcinogenesis. 2010. PMID: 20176656

3

Intestinal and extra-intestinal tumor multiplicities in the Apc1638N mouse model after exposure to X-rays.

van der Houven van Oordt CW, Smits R, Williamson SL, Luz A, Khan PM, Fodde R, van der Eb AJ, Breuer ML. van der Houven van Oordt CW, et al. Among authors: smits r. Carcinogenesis. 1997 Nov;18(11):2197-203. doi: 10.1093/carcin/18.11.2197. Carcinogenesis. 1997. PMID: 9395221

4

Apc1638N: a mouse model for familial adenomatous polyposis-associated desmoid tumors and cutaneous cysts.

Smits R, van der Houven van Oordt W, Luz A, Zurcher C, Jagmohan-Changur S, Breukel C, Khan PM, Fodde R. Smits R, et al. Gastroenterology. 1998 Feb;114(2):275-83. doi: 10.1016/s0016-5085(98)70478-0. Gastroenterology. 1998. PMID: 9453487

5

Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity.

Smits R, Hofland N, Edelmann W, Geugien M, Jagmohan-Changur S, Albuquerque C, Breukel C, Kucherlapati R, Kielman MF, Fodde R. Smits R, et al. Genes Chromosomes Cancer. 2000 Nov;29(3):229-39. doi: 10.1002/1098-2264(2000)9999:9999<::aid-gcc1033>3.0.co;2-r. Genes Chromosomes Cancer. 2000. PMID: 10992298

6

E-cadherin and adenomatous polyposis coli mutations are synergistic in intestinal tumor initiation in mice.

Smits R, Ruiz P, Diaz-Cano S, Luz A, Jagmohan-Changur S, Breukel C, Birchmeier C, Birchmeier W, Fodde R. Smits R, et al. Gastroenterology. 2000 Oct;119(4):1045-53. doi: 10.1053/gast.2000.18162. Gastroenterology. 2000. PMID: 11040191

7

Disease model: familial adenomatous polyposis.

Fodde R, Smits R. Fodde R, et al. Among authors: smits r. Trends Mol Med. 2001 Aug;7(8):369-73. doi: 10.1016/s1471-4914(01)02050-0. Trends Mol Med. 2001. PMID: 11516998 Review.

8

Serrated adenomas and mixed polyposis caused by a splice acceptor deletion in the mouse Smad4 gene.

Hohenstein P, Molenaar L, Elsinga J, Morreau H, van der Klift H, Struijk A, Jagmohan-Changur S, Smits R, van Kranen H, van Ommen GJ, Cornelisse C, Devilee P, Fodde R. Hohenstein P, et al. Among authors: smits r. Genes Chromosomes Cancer. 2003 Mar;36(3):273-82. doi: 10.1002/gcc.10169. Genes Chromosomes Cancer. 2003. PMID: 12557227

9

Apc1638T: a mouse model delineating critical domains of the adenomatous polyposis coli protein involved in tumorigenesis and development.

Smits R, Kielman MF, Breukel C, Zurcher C, Neufeld K, Jagmohan-Changur S, Hofland N, van Dijk J, White R, Edelmann W, Kucherlapati R, Khan PM, Fodde R. Smits R, et al. Genes Dev. 1999 May 15;13(10):1309-21. doi: 10.1101/gad.13.10.1309. Genes Dev. 1999. PMID: 10346819 Free PMC article.

10

The genetic background modifies the spontaneous and X-ray-induced tumor spectrum in the Apc1638N mouse model.

van der Houven van Oordt CW, Smits R, Schouten TG, Houwing-Duistermaat JJ, Williamson SL, Luz A, Meera Khan P, van der Eb AJ, Breuer ML, Fodde R. van der Houven van Oordt CW, et al. Among authors: smits r. Genes Chromosomes Cancer. 1999 Mar;24(3):191-8. doi: 10.1002/(sici)1098-2264(199903)24:3<191::aid-gcc3>3.0.co;2-l. Genes Chromosomes Cancer. 1999. PMID: 10451698

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