Watnick TJ - Search Results

1

Polycystin: in vitro synthesis, in vivo tissue expression, and subcellular localization identifies a large membrane-associated protein.

Ibraghimov-Beskrovnaya O, Dackowski WR, Foggensteiner L, Coleman N, Thiru S, Petry LR, Burn TC, Connors TD, Van Raay T, Bradley J, Qian F, Onuchic LF, Watnick TJ, Piontek K, Hakim RM, Landes GM, Germino GG, Sandford R, Klinger KW. Ibraghimov-Beskrovnaya O, et al. Among authors: watnick tj. Proc Natl Acad Sci U S A. 1997 Jun 10;94(12):6397-402. doi: 10.1073/pnas.94.12.6397. Proc Natl Acad Sci U S A. 1997. PMID: 9177229 Free PMC article.

2

Tolvaptan in Later-Stage Autosomal Dominant Polycystic Kidney Disease.

Torres VE, Chapman AB, Devuyst O, Gansevoort RT, Perrone RD, Koch G, Ouyang J, McQuade RD, Blais JD, Czerwiec FS, Sergeyeva O; REPRISE Trial Investigators. Torres VE, et al. N Engl J Med. 2017 Nov 16;377(20):1930-1942. doi: 10.1056/NEJMoa1710030. Epub 2017 Nov 4. N Engl J Med. 2017. PMID: 29105594 Free article. Clinical Trial.

3

Polycystin-2-dependent transcriptome reveals early response of autosomal dominant polycystic kidney disease.

Jung HJ, Dixon EE, Coleman R, Watnick T, Reiter JF, Outeda P, Cebotaru V, Woodward OM, Welling PA. Jung HJ, et al. Physiol Genomics. 2023 Nov 1;55(11):565-577. doi: 10.1152/physiolgenomics.00040.2023. Epub 2023 Sep 18. Physiol Genomics. 2023. PMID: 37720991 Free PMC article.

4

Vascular Complications in Autosomal Dominant Polycystic Kidney Disease: Perspectives, Paradigms, and Current State of Play.

Gulati A, Watnick T. Gulati A, et al. Adv Kidney Dis Health. 2023 Sep;30(5):429-439. doi: 10.1053/j.akdh.2023.09.004. Adv Kidney Dis Health. 2023. PMID: 38097333 Review.

5

The molecular basis of focal cyst formation in human autosomal dominant polycystic kidney disease type I.

Qian F, Watnick TJ, Onuchic LF, Germino GG. Qian F, et al. Among authors: watnick tj. Cell. 1996 Dec 13;87(6):979-87. doi: 10.1016/s0092-8674(00)81793-6. Cell. 1996. PMID: 8978603 Free article.

6

An unusual pattern of mutation in the duplicated portion of PKD1 is revealed by use of a novel strategy for mutation detection.

Watnick TJ, Piontek KB, Cordal TM, Weber H, Gandolph MA, Qian F, Lens XM, Neumann HP, Germino GG. Watnick TJ, et al. Hum Mol Genet. 1997 Sep;6(9):1473-81. doi: 10.1093/hmg/6.9.1473. Hum Mol Genet. 1997. PMID: 9285784

7

Gene conversion is a likely cause of mutation in PKD1.

Watnick TJ, Gandolph MA, Weber H, Neumann HP, Germino GG. Watnick TJ, et al. Hum Mol Genet. 1998 Aug;7(8):1239-43. doi: 10.1093/hmg/7.8.1239. Hum Mol Genet. 1998. PMID: 9668165

8

Somatic mutation in individual liver cysts supports a two-hit model of cystogenesis in autosomal dominant polycystic kidney disease.

Watnick TJ, Torres VE, Gandolph MA, Qian F, Onuchic LF, Klinger KW, Landes G, Germino GG. Watnick TJ, et al. Mol Cell. 1998 Aug;2(2):247-51. doi: 10.1016/s1097-2765(00)80135-5. Mol Cell. 1998. PMID: 9734362 Free article.

9

Somatic PKD2 mutations in individual kidney and liver cysts support a "two-hit" model of cystogenesis in type 2 autosomal dominant polycystic kidney disease.

Pei Y, Watnick T, He N, Wang K, Liang Y, Parfrey P, Germino G, St George-Hyslop P. Pei Y, et al. J Am Soc Nephrol. 1999 Jul;10(7):1524-9. doi: 10.1681/ASN.V1071524. J Am Soc Nephrol. 1999. PMID: 10405208

10

Molecular basis of autosomal dominant polycystic kidney disease.

Watnick T, Germino GG. Watnick T, et al. Semin Nephrol. 1999 Jul;19(4):327-43. Semin Nephrol. 1999. PMID: 10435671 Review.

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