F31 NS101762/NS/NINDS NIH HHS/United States[Grants and Funding] - Search Results

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Year	Number of Results
2019	1
2020	1
2021	2
2024	0

Page 1

Temporal manipulation of Cdkl5 reveals essential postdevelopmental functions and reversible CDKL5 deficiency disorder-related deficits.

Terzic B, Davatolhagh MF, Ho Y, Tang S, Liu YT, Xia Z, Cui Y, Fuccillo MV, Zhou Z. Terzic B, et al. J Clin Invest. 2021 Oct 15;131(20):e143655. doi: 10.1172/JCI143655. J Clin Invest. 2021. PMID: 34651584 Free PMC article.

Altered NMDAR signaling underlies autistic-like features in mouse models of CDKL5 deficiency disorder.

Tang S, Terzic B, Wang IJ, Sarmiento N, Sizov K, Cui Y, Takano H, Marsh ED, Zhou Z, Coulter DA. Tang S, et al. Nat Commun. 2019 Jun 14;10(1):2655. doi: 10.1038/s41467-019-10689-w. Nat Commun. 2019. PMID: 31201320 Free PMC article.

X-linked cellular mosaicism underlies age-dependent occurrence of seizure-like events in mouse models of CDKL5 deficiency disorder.

Terzic B, Cui Y, Edmondson AC, Tang S, Sarmiento N, Zaitseva D, Marsh ED, Coulter DA, Zhou Z. Terzic B, et al. Neurobiol Dis. 2021 Jan;148:105176. doi: 10.1016/j.nbd.2020.105176. Epub 2020 Nov 13. Neurobiol Dis. 2021. PMID: 33197557 Free PMC article.

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