R03CA165122/CA/NCI NIH HHS/United States[Grants and Funding] - Search Results

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2013	1
2015	1
2016	2
2024	0

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Co-activation of AKT and c-Met triggers rapid hepatocellular carcinoma development via the mTORC1/FASN pathway in mice.

Hu J, Che L, Li L, Pilo MG, Cigliano A, Ribback S, Li X, Latte G, Mela M, Evert M, Dombrowski F, Zheng G, Chen X, Calvisi DF. Hu J, et al. Sci Rep. 2016 Feb 9;6:20484. doi: 10.1038/srep20484. Sci Rep. 2016. PMID: 26857837 Free PMC article.

Inactivation of fatty acid synthase impairs hepatocarcinogenesis driven by AKT in mice and humans.

Li L, Pilo GM, Li X, Cigliano A, Latte G, Che L, Joseph C, Mela M, Wang C, Jiang L, Ribback S, Simile MM, Pascale RM, Dombrowski F, Evert M, Semenkovich CF, Chen X, Calvisi DF. Li L, et al. J Hepatol. 2016 Feb;64(2):333-341. doi: 10.1016/j.jhep.2015.10.004. Epub 2015 Oct 22. J Hepatol. 2016. PMID: 26476289 Free PMC article.

SCD1 Expression is dispensable for hepatocarcinogenesis induced by AKT and Ras oncogenes in mice.

Li L, Wang C, Calvisi DF, Evert M, Pilo MG, Jiang L, Yuneva M, Chen X. Li L, et al. PLoS One. 2013 Sep 19;8(9):e75104. doi: 10.1371/journal.pone.0075104. eCollection 2013. PLoS One. 2013. PMID: 24069385 Free PMC article.

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