R21 EY035844/EY/NEI NIH HHS/United States[Grants and Funding] - Search Results

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REDD1 Is a Promising Therapeutic Target to Combat the Development of Diabetes Complications: A Report on Research Supported by Pathway to Stop Diabetes.

Sunilkumar S, Dennis MD. Sunilkumar S, et al. Diabetes. 2024 Oct 1;73(10):1553-1562. doi: 10.2337/dbi24-0013. Diabetes. 2024. PMID: 38976480 Free PMC article. Review.

NLRP3 Inflammasome Priming in the Retina of Diabetic Mice Requires REDD1-Dependent Activation of GSK3β.

McCurry CM, Sunilkumar S, Subrahmanian SM, Yerlikaya EI, Toro AL, VanCleave AM, Stevens SA, Barber AJ, Sundstrom JM, Dennis MD. McCurry CM, et al. Invest Ophthalmol Vis Sci. 2024 Mar 5;65(3):34. doi: 10.1167/iovs.65.3.34. Invest Ophthalmol Vis Sci. 2024. PMID: 38546584 Free PMC article.

Deletion of the stress response protein REDD1 prevents sodium iodate-induced RPE damage and photoreceptor loss.

Subrahmanian SM, Yerlikaya EI, Sunilkumar S, Toro AL, McCurry CM, Grillo SL, Barber AJ, Sundstrom JM, Dennis MD. Subrahmanian SM, et al. Geroscience. 2024 Oct 5. doi: 10.1007/s11357-024-01362-2. Online ahead of print. Geroscience. 2024. PMID: 39367169

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